Increased endothelial cell mobility following stabilization of hypoxia-inducible factor-steps towards therapeutic angiogenesis

Increased endothelial cell mobility following stabilization of hypoxia-inducible factor-steps towards therapeutic angiogenesis

Bibliográfalaš dieđut
Váldodahkkit:	Kelly, D, Mecinovic, J, Chitkara, K, Schofield, C, Gershlick, A
Materiálatiipa:	Conference item
Almmustuhtton:	2008

Geahča maid

Evidence for a potential new method of therapeutic angiogenesis: Increased endothelial cell mobility and tubule formation in cell culture after stabilisation of hypoxia-inducible factor
Dahkki: Kelly, D, et al.
Almmustuhtton: (2008)

Enhanced Endothelial Tubule Formation in Response to Proly-Hydroxylase Inhibitor FG-2216: A Step Towards Therapeutic Angiogenesis in Chronic Total Occlusions
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In-vivo observations on angiogenesis: Dose-dependent effects of the prolyl-4-hydroxylase inhibitor, di-methyl oxalyl-glycine, on arterial development in embryonic zebrafish
Dahkki: Kelly, D, et al.
Almmustuhtton: (2008)

In-vivo observations on angiogenesis in embryonic zebrafish: dose-dependent effects of the prolyl-4-hydroxylase inhibitor, Di-methyl oxalyl-glycine, on arterial development
Dahkki: Kelly, D, et al.
Almmustuhtton: (2008)

74 Pro-Angiogenic Effects of the Prolyl-hydroxylase Inhibitor FG-2216/BIQ: Therapeutic Angiogenesis for the Treatment of Chronic Total Occlusions.
Dahkki: Barnett, G, et al.
Almmustuhtton: (2014)

UPREGULATION OF HYPDXIA-INDUCIBLE FACTOR 1 alpha BY DI-METHYL OXALYL GLYCINE PROMOTES ANGIOGENESIS WITHIN ISCHAEMIC MYOCARDIUM IN A PORCINE CORONARY OCCLUSION MODEL
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Almmustuhtton: (2010)

Pro-angiogenic effects of proly-hydroxylase inhibitor FG-2216 and potential for use in a novel strategy of therapeutic angiogenesis for difficult cases of coronary total occlusion
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Almmustuhtton: (2003)

Hypoxia-Induced miR-210 Promotes Endothelial Cell Permeability and Angiogenesis via Exosomes in Pancreatic Ductal Adenocarcinoma
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Vascular endothelial growth factor C is increased in endometrium and promotes endothelial functions, vascular permeability and angiogenesis and growth of endometriosis.
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Almmustuhtton: (2013)

Vascular endothelial growth factor C is increased in endometrium and promotes endothelial functions, vascular permeability and angiogenesis and growth of endometriosis
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Differential Effects of Leptin and Adiponectin in Endothelial Angiogenesis
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Almmustuhtton: (2015-01-01)

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Cavin-2 regulates the activity and stability of endothelial nitric-oxide synthase (eNOS) in angiogenesis
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Hypoxia upregulates angiogenesis and synovial cell migration in rheumatoid arthritis.
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Almmustuhtton: (2004)

Hypoxia-induced P4HA1 overexpression promotes post-ischemic angiogenesis by enhancing endothelial glycolysis through downregulating FBP1
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Endothelial cell senescence contributes to pathological retinal angiogenesis
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Almmustuhtton: (2024-10-01)

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Advances in endothelial cell lipid metabolism and tumor angiogenesis
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Hypoxia suppresses glucose-induced increases in collective cell migration in vascular endothelial cell monolayers
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Principles of targeting endothelial cell metabolism to treat angiogenesis and endothelial cell dysfunction in disease
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Therapeutic manipulation of the HIF hydroxylases.
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Almmustuhtton: (2010)

Unveiling the Regulatory Role of LncRNA MYU in Hypoxia-Induced Angiogenesis via the miR-23a-3p Axis in Endothelial Cells
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A role of Agrin in maintaining the stability of vascular endothelial growth factor receptor-2 during tumor angiogenesis
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Epigenetic regulation of angiogenesis and its therapeutics
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Almmustuhtton: (2012)

Fbxw7 controls angiogenesis by regulating endothelial Notch activity.
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