Molecular and cellular effects of oncogene cooperation in a genetically accurate AML mouse model.
Biallelic CEBPA mutations and FMS-like tyrosine kinase receptor 3 (FLT3) length mutations are frequently identified in human acute myeloid leukemia (AML) with normal cytogenetics. However, the molecular and cellular mechanisms of oncogene cooperation remain unclear because of a lack of disease model...
Main Authors: | Reckzeh, K, Bereshchenko, O, Mead, A, Rehn, M, Kharazi, S, Jacobsen, SE, Nerlov, C, Cammenga, J |
---|---|
Formato: | Journal article |
Idioma: | English |
Publicado em: |
2012
|
Registos relacionados
-
Molecular and cellular effects of oncogene cooperation in a genetically accurate AML mouse model.
Por: Reckzeh, K, et al.
Publicado em: (2012) -
ROLE OF BIALLELIC C/EBPALPHA MUTATIONS AND FLT-3 ITD IN INITIATION AND MAINTENANCE OF AML
Por: Reckzeh, K, et al.
Publicado em: (2010) -
Hematopoietic stem cell expansion precedes the generation of committed myeloid leukemia-initiating cells in C/EBPalpha mutant AML.
Por: Bereshchenko, O, et al.
Publicado em: (2009) -
MEIS2 Is an Oncogenic Partner in AML1-ETO-Positive AML
Por: Naidu M. Vegi, et al.
Publicado em: (2016-07-01) -
Germline counterparts of oncogenic mutations: who gives a JAK?.
Por: Mead, A, et al.
Publicado em: (2013)