Association of superoxide production by NAD(P)H oxidase with systaemic endothelial dysfunction in human blood vessels: relationship to clinical risk factors

Association of superoxide production by NAD(P)H oxidase with systaemic endothelial dysfunction in human blood vessels: relationship to clinical risk factors

מידע ביבליוגרפי
Main Authors:	Guzik, T, West, N, Black, E, McDonald, D, Ratnatunga, C, Pillai, R, Channon, K
פורמט:	Journal article
יצא לאור:	2000

פריטים דומים

Vascular superoxide production by NAD(P)H oxidase: association with endothelial dysfunction and clinical risk factors.
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Superoxide production by NAD(P)H oxidase in human blood vessels: Functional interaction with nitric oxide
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Relationship of NAD(P)H oxidase activity in human blood vessels to endothelial dysfunction and clinical risk factors
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UltraRapid communications : vascular superoxide production by NAD(P)H OxidaseAssociation with endothelial dysfunction and clinical risk factors
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Superoxide generation by NAD(P)H oxidase in human sphenous vein: Interaction with nitric oxide
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Functional impact of genetic polymorphisms in NAD(P)H oxidase p22phox subunit on vascular superoxide production in atherosclerosis
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Functional effect of the C242T polymorphism in the NAD(P)H oxidase p22phox gene on vascular superoxide production in atherosclerosis.
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Functional importance of C242T polymorphism in NAD(P)H oxidase p22phox subunit on vascular superoxide production in atherosclerosis
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Enhanced superoxide anion production in experimental venous bypass grafts is mediated by NAD(P)H oxidase
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Vascular superoxide generation by NAD(P)H oxidase is increased in humans with type II diabetes mellitus
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Mechanisms of increased vascular superoxide production in human diabetes mellitus: role of NAD(P)H oxidase and endothelial nitric oxide synthase.
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Superoxide generation by NAD(P)H oxidases in human bypass graft vessels.
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Enhanced superoxide production in experimental venous bypass graft intimal hyperplasia: role of NAD(P)H oxidase.
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Systemic upregulation of vascular NAD(P)H oxidase in human atherosclerosis: Association with clinical risk factors.
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Mechanisms of superoxide production in human blood vessels: relationship to endothelial dysfunction, clinical and genetic risk factors.
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Nitric oxide-superoxide interactions in human blood vessels: implications for vascular peroxynitrite formation
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Nitric oxide modulates superoxide release and peroxynitrite formation in human blood vessels.
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Superoxide generation during cardiopulmonary bypass in conventional coronary artery bypass graft surgery: is there a role for myocardial NAD(P)H oxidase?
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Relationship between the G894T polymorphism (Glu298Asp variant) in endothelial nitric oxide synthase and nitric oxide-mediated endothelial function in human atherosclerosis.
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A myocardial nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation (AF)
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Direct improvement of endothelial function and superoxide production in human vessels, by oral folic acid treatment: A double blind placebo controlled study
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Relationships of promoter and coding sequence genetic polymorphisms of p22phox subunit to the vascular NAD(P)H oxidase activity
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A myocardial Nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation
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A myocardial Nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation.
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5-methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production in vessels from patients with coronary artery disease
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Endothelial dysfunction and increased vascular superoxide generation in the type II diabetic (Goto-Kakisaki) rat occur independently of plasma cholesterol levels
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Role of NAD(P)H oxidase in superoxide generation and endothelial dysfunction in Goto-Kakizaki (GK) rats as a model of nonobese NIDDM.
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Chronic treatment with folic acid improves endothelial function and decreases superoxide production in human vessels. A double blind placebo control study
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Minimally invasive saphenous vein harvesting: effects on endothelial and smooth muscle function.
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Low- and high- dose treatment with folic acid, equally improve endothelial function and decrease superoxide production in human vessels via tetrahydrobiopterin-mediated eNOS coupling
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Endothelial and smooth muscle function are preserved with minimally invasive saphenous vein harvest: A prospective randomised trial.
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5-Methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production by improving eNOS coupling and increasing tetrahydrobiopterin bioavailability in human vessels in vivo
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Atrial NAD(P)H oxidase activity predicts the development of atrial fibrillation after coronary artery bypass graft surgery
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5-methyltetrahydrofolate rapidly improves endothelial function and decreases superoxide production in human vessels: effects on vascular tetrahydrobiopterin availability and endothelial nitric oxide synthase coupling.
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Atrial NAD(P)H oxidase activity predicts the development of atrial fibrillation after on-pump coronary artery bypass graft surgery
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Modification of superoxide-nitric oxide interactions in experimental venous bypass graft pathobiology by NOS gene transfer
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A myocardial nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation
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Systemic regulation of vascular NAD(P)H oxidase activity and nox isoform expression in human arteries and veins
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Systemic regulation of vascular NAD(P)H oxidase activity and nox isoform expression in human arteries and veins.
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