Characterisation of distinct GATA-1 complexes in erythroid cells reveals a major role in gene repression.

Characterisation of distinct GATA-1 complexes in erythroid cells reveals a major role in gene repression.

Bibliografski detalji
Glavni autori:	Rodriguez, P, Bonte, E, Krijgsveld, J, Kolodziej, K, Guyot, B, Vyas, P, de Boer, E, Grosveld, F, Strouboulis, J
Format:	Conference item
Izdano:	2005

Slični predmeti

GATA-1 forms distinct activating and repressive complexes in erythroid cells
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GATA-1 forms distinct activating and repressive complexes in erythroid cells.
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Characterization of distinct GATA-1 complexes in erythroid cells.
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Characterisation erythroid-specific cis-elements regulating the key transcription factor GATA1.
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Histone demethylase LSD1-mediated repression of GATA-2 is critical for erythroid differentiation
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GATA1 and PU.1 Bind to Ribosomal Protein Genes in Erythroid Cells: Implications for Ribosomopathies.
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The bHLH protein SCL/Tal-l interacts with the co-repressor ETO-2 in erythroid cells and megakaryocytes.
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The oncoprotein SCL/Tal-1 associates with the co-repressor ETO-2 in multiprotein complexes in erythroid cells and megalkaryocytes.
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Lineage-specific combinatorial action of enhancers regulates mouse erythroid Gata1 expression.
od: Drissen, R, i dr.
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An intrinsic but cell-nonautonomous defect in GATA-1-overexpressing mouse erythroid cells.
od: Whyatt, D, i dr.
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Characterisation and comparison of the human and mouse GATA-1 transcriptional domain.
od: Vyas, P, i dr.
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Deletion of the major GATA1 enhancer HS 1 does not affect eosinophil GATA1 expression and eosinophil differentiation.
od: Guyot, B, i dr.
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Characterization of megakaryocyte GATA1-interacting proteins: the corepressor ETO2 and GATA1 interact to regulate terminal megakaryocyte maturation.
od: Hamlett, I, i dr.
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ETO-2 associates with SCL in erythroid cells and megakaryocytes and provides repressor functions in erythropoiesis.
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GATA-1 genome-wide occupancy associates with distinct epigenetic profiles in mouse fetal liver erythropoiesis.
od: Papadopoulos, G, i dr.
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Dynamics of GATA1 binding and expression response in a GATA1-induced erythroid differentiation system
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A novel complex, RUNX1-MYEF2, represses hematopoietic genes in erythroid cells.
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C/EBPα and GATA-2 mutations induce bilineage acute erythroid leukemia through transformation of a neomorphic neutrophil-erythroid progenitor
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Determinants of GATA1-mediated gene regulation during erythroid maturation
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TP53 Modulates Oxidative Stress in Gata1+ Erythroid Cells
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Dynamics of the Epigenetic Landscape During Erythroid Differentiation after Gata1 Restoration
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FOG-1 and GATA-1 act sequentially to specify definitive megakaryocytic and erythroid progenitors
od: Mancini, E, i dr.
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FOG-1 and GATA-1 act sequentially to specify definitive megakaryocytic and erythroid progenitors.
od: Mancini, E, i dr.
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Impact of mutant haemopoietic transcription factor GATA1s on haemopoiesis
od: Vyas, P, i dr.
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Deubiquitylase USP7 regulates human terminal erythroid differentiation by stabilizing GATA1
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Hematopoietic regulation of transcription factor GATA1 and mutations causing hereditary erythroid and megakaryocytic disorders
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The Pleiotropic Effects of GATA1 and KLF1 in Physiological Erythropoiesis and in Dyserythropoietic Disorders
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Single-cell profiling of human megakaryocyte-erythroid progenitors identifies distinct megakaryocyte and erythroid differentiation pathways
od: Psaila, B, i dr.
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GATA1 mutation analysis demonstrates two distinct primary leukemias in a child with down syndrome; implications for leukemogenesis.
od: Hellebostad, M, i dr.
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Global transcriptome and chromatin occupancy analysis reveal the short isoform of GATA1 is deficient for erythroid specification and gene expression
od: Timothy M. Chlon, i dr.
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A single cis element maintains repression of the key developmental regulator Gata2.
od: Jonathan W Snow, i dr.
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Transcriptional repression of Gata3 is essential for early B cell commitment.
od: Banerjee, A, i dr.
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S160: GATA2B HAPLOINSUFFICIENCY CAUSES ABERRANT TRANSCRIPTIONAL REGULATORY SIGNATURES IN HSPCS RESULTING IN MYELOID AND ERYTHROID DYSPLASIA IN ZEBRAFISH
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The <it>GATA1s</it> isoform is normally down-regulated during terminal haematopoietic differentiation and over-expression leads to failure to repress <it>MYB, CCND2</it> and <it>SKI</it> during erythroid differentiation of K562 cells
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hGATA1 Under the Control of a μLCR/β-Globin Promoter Rescues the Erythroid but Not the Megakaryocytic Phenotype Induced by the Gata1low Mutation in Mice
od: Fabrizio Martelli, i dr.
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Novel binding partners of Ldb1 are required for haematopoietic development.
od: Meier, N, i dr.
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Novel binding partners of Ldb1 are required for haematopoietic development
od: Meier, N, i dr.
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Acute erythroid leukemias have a distinct molecular hierarchy from non-erythroid acute myeloid leukemias
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