Cyclic AMP stimulates exocytosis in mouse B-cells by both PKA-dependent and PKA-independent mechanisms

Cyclic AMP stimulates exocytosis in mouse B-cells by both PKA-dependent and PKA-independent mechanisms

Bibliografiske detaljer
Main Authors:	Eliasson, L, Renstrom, E, Ammala, C, Rorsman, P
Format:	Journal article
Udgivet:	1996

Lignende værker

REGULATION OF EXOCYTOSIS IN THE PANCREATIC B-CELL - INTERACTIONS BETWEEN PKA AND PKC
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Protein kinase A-dependent and -independent stimulation of exocytosis by cAMP in mouse pancreatic B-cells.
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ATP-dependence of exocytosis in mouse pancreatic beta-cells
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SUR1 regulates PKA-independent cAMP-induced granule priming in mouse pancreatic B-cells.
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Ca(2+)- and GTP-dependent exocytosis in mouse pancreatic beta-cells involves both common and distinct steps.
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RELATIONSHIP BETWEEN EXOCYTOSIS AND ENDOCYTOSIS IN PANCREATIC B-CELLS
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EXOCYTOSIS IN MOUSE PANCREATIC B-CELLS IS REGULATED BY THE BALANCE BETWEEN PHOSPHORYLATION AND DEPHOSPHORYLATION
af: Rorsman, P, et al.
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CONTROL OF EXOCYTOSIS WITH B-CELL AT PROXIMAL AND DISTAL LEVELS OF THE SECRETORY CASCADE
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TOLBUTAMIDE STIMULATES EXOCYTOSIS BY DIRECT INTERACTION WITH THE SECRETORY MACHINERY IN B-CELLS
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Acetylcholine stimulates exocytosis by acceleration of granula mobilisation in mouse pancreatic B-cells
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Cyclic AMP-dependent PKA phosphorylates starfish sperm proteins during acrosome reaction
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Correlation between functional and ultrastructual measurements of exocytosis in mouse pancreatic B-cells
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Co-localization of L-type Ca2+ channels and insulin-containing secretory granules and its significance for the initiation of exocytosis in mouse pancreatic B-cells.
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Intracellular tolbutamide stimulates calcium-dependent exocytosis in pancreatic B-cells
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Cyclic AMP stimulates insulin exocytosis by modification of granules already present at the release sites
af: Renstrom, E, et al.
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CaM kinase II-dependent mobilization of secretory granules underlies acetylcholine-induced stimulation of exocytosis in mouse pancreatic B-cells.
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Cooling inhibits exocytosis in single mouse pancreatic B-cells by suppression of granule mobilization.
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DIRECT DEMONSTRATION THAT CAMP POTENTIATES CALCIUM-DEPENDENT EXOCYTOSIS IN SINGLE B-CELLS
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PKC-dependent stimulation of exocytosis by sulfonylureas in pancreatic beta cells.
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Rapid ATP-dependent priming of secretory granules precedes Ca(2+)-induced exocytosis in mouse pancreatic B-cells.
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ApoA-I Milano stimulates lipolysis in adipose cells independently of cAMP/PKA activation
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Exocytosis elicited by action potentials and voltage-clamp calcium currents in individual mouse pancreatic B-cells.
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Cyclic AMP Regulates Key Features of Macrophages via PKA: Recruitment, Reprogramming and Efferocytosis
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SULFONYLUREA AND EXOCYTOSIS IN MOUSE BETA-CELLS - A POSSIBLE INTERACTION WITH PROTEIN-KINASE-C
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Adrenaline stimulates secretion in rat pancreatic alpha-cells by PKA-dependent acceleration of Ca2+ influx through 1-type Ca2+- channels
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Exocytosis depends on granular chloride fluxes regulated by a sulfonylurea receptor-like protein in insulin-secreting B-cells
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Noradrenalin and dopamine receptors both control cAMP-PKA signaling throughout the cerebral cortex
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Regulation of Cardiac PKA Signaling by cAMP and Oxidants
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Evolution of the cAMP-dependent protein kinase (PKA) catalytic subunit isoforms.
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Evolutionary paths of the cAMP-dependent protein kinase (PKA) catalytic subunits.
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Calcium-independent potentiation of insulin release by cyclic AMP in single beta-cells.
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Nobiletin Stimulates Chloride Secretion in Human Bronchial Epithelia via a cAMP/PKA-Dependent Pathway
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Cpk2, a Catalytic Subunit of Cyclic AMP-PKA, Regulates Growth and Pathogenesis in Rice Blast
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Cpk2, a Catalytic Subunit of Cyclic AMP-PKA, Regulates Growth and Pathogenesis in Rice Blast
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Modulation of HERG K+ Channels by Chronic Exposure to Activators and Inhibitors of PKA and PKC: Actions Independent of PKA and PKC Phosphorylation
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Nateglinide, but not repaglinide, stimulates growth hormone release in rat pituitary cells by inhibition of K channels and stimulation of cyclic AMP-dependent exocytosis.
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EXOCYTOSIS INDUCED BY A SINGLE ACTION-POTENTIAL IN INSULIN-SECRETING B-CELLS
af: Ammala, C, et al.
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Sulfonylurea-mediated stimulation of insulin exocytosis via an ATP-sensitive K+ channel-independent action.
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Plasmodium falciparum regulatory subunit of cAMP-dependent PKA and anion channel conductance.
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Cardiac cAMP-PKA Signaling Compartmentalization in Myocardial Infarction
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