Effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease

Manylion Llyfryddiaeth
Prif Awduron:	Antoniades, C, Shirodaria, C, Leeson, P, Lee, J, Warrick, N, Taggart, D, Ratnatunga, C, Pillai, R, Channon, K
Fformat:	Conference item
Cyhoeddwyd:	2006

Rapid effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

Direct effects of 5-methyl-tetrahydrofolate on vascular endotheliurn and superoxide production in human arteries and veins
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

5-methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production in vessels from patients with coronary artery disease
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

5-Methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production by improving eNOS coupling and increasing tetrahydrobiopterin bioavailability in human vessels in vivo
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

5-Methyl-tetrahydrofolic acid rapidly improves endothelial function and superoxide production in human atherosclerosis: Effects on tetrahyd robiopterin-dependent eNOS coupling
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

5-methyl-tetrahydrofolate increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling and preventing the intracellular oxidation of tetrahy-drobiopterin, in human vessels in vivo
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
gan: Antoniades, C, et al.
Cyhoeddwyd: (2009)

Genetic Variability of Methyl-Tetrahydrofolate-Reductase Modifies eNOS Coupling in Human Arteries and Veins: Effects on Vascular Superoxide Generation and Nitric Oxide Bioavailabllity
gan: Antoniades, C, et al.
Cyhoeddwyd: (2008)

5-methyl-tetrahydrofolate increases eNOS activity by preventing peroxynitrite-induced oxidation of tetrahydrobiopterin in human vessels
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

Intravenous infusion of 5-methyl-tetrahydrofolate rapidly improves eNOS Coupling. In human vessels by increasing tetrahydrobiopterin bioavailability
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

Vascular Homocysteine Versus 5-Methyl-Tetrahydrofolate As Regulators of Vascular Redox and Endothelial Function in Human Vessels
gan: Antoniades, C, et al.
Cyhoeddwyd: (2009)

Vascular Homocysteine vs 5-Methyl-Tetrahydrofolate as Regulators of Vascular Redox and Endothelial Function in Human Vessels
gan: Antoniades, C, et al.
Cyhoeddwyd: (2008)

Association of plasma asymmetrical dimethylarginine (ADMA) with elevated vascular superoxide production and endothelial nitric oxide synthase uncoupling: implications for endothelial function in human atherosclerosis.
gan: Antoniades, C, et al.
Cyhoeddwyd: (2009)

5-methyltetrahydrofolate rapidly improves endothelial function and decreases superoxide production in human vessels: effects on vascular tetrahydrobiopterin availability and endothelial nitric oxide synthase coupling.
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

Pre-operative flow mediated dilatation in patients undergoing coronary artery bypass surgery: Prediction of NO-bioavailability and vascular superoxide production in arterial and vein grafts
gan: Antoniades, C, et al.
Cyhoeddwyd: (2008)

Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
gan: Antoniades, C, et al.
Cyhoeddwyd: (2008)

Direct improvement of endothelial function and superoxide production in human vessels, by oral folic acid treatment: A double blind placebo controlled study
gan: Antoniades, C, et al.
Cyhoeddwyd: (2005)

GCH1 haplotype determines vascular and plasma biopterin availability in coronary artery disease effects on vascular superoxide production and endothelial function.
gan: Antoniades, C, et al.
Cyhoeddwyd: (2008)

The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
gan: Antoniades, C, et al.
Cyhoeddwyd: (2007)

Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels
gan: Antoniades, C, et al.
Cyhoeddwyd: (2007)

Low- and high- dose treatment with folic acid, equally improve endothelial function and decrease superoxide production in human vessels via tetrahydrobiopterin-mediated eNOS coupling
gan: Antoniades, C, et al.
Cyhoeddwyd: (2007)

Chronic treatment with folic acid improves endothelial function and decreases superoxide production in human vessels. A double blind placebo control study
gan: Shirodaria, C, et al.
Cyhoeddwyd: (2005)

Relationships between vascular/plasma biopterins and endothelial nitric oxide synthase coupling, endothelial function and inflammation, in human atherosclerosis
gan: Antoniades, C, et al.
Cyhoeddwyd: (2008)

Soluble CD40-ligand levels are associated with vascular superoxide production and eNOS coupling in human vessels: Interrelationships with homocysteine biosynthetic pathway
gan: Antoniades, C, et al.
Cyhoeddwyd: (2008)

No additional improvement in vascular function with high dose vs. low dose folic acid in coronary artery disease - Implications for HOPE-2
gan: Shirodaria, C, et al.
Cyhoeddwyd: (2006)

Preoperative evaluation of flow-mediated dilatation in the left radial artery or the right brachial artery, as a predictor of nitric-oxide bioavailability in coronary artery bypass grafts
gan: Antoniades, C, et al.
Cyhoeddwyd: (2006)

Global improvements in both central and peripheral vascular function after short-term folic acid treatment in patients with coronary artery disease - A cardiovascular MR study
gan: Shirodaria, C, et al.
Cyhoeddwyd: (2005)

Altered plasma vs. vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function and inflammation
gan: Antomades, C, et al.
Cyhoeddwyd: (2007)

Chronic treatment with folic acid improves nitric oxide bioavailability and decreases superoxide production in human vessels, through tetrahydrobiopterin-mediated improvement of eNOS coupling
gan: Antoniades, C, et al.
Cyhoeddwyd: (2007)

Short-term folic acid treatment improves central and peripheral vascular function in patients with coronary artery disease - A cardiovascular magnetic resonance study
gan: Shirodaria, C, et al.
Cyhoeddwyd: (2006)

Vascular superoxide production by NAD(P)H oxidase: association with endothelial dysfunction and clinical risk factors.
gan: Guzik, T, et al.
Cyhoeddwyd: (2000)

Global improvement of vascular function and redox state with low-dose folic acid: implications for folate therapy in patients with coronary artery disease.
gan: Shirodaria, C, et al.
Cyhoeddwyd: (2007)

Relation of preoperative radial artery flow-mediated dilatation to nitric oxide bioavailability in radial artery grafts used in off-pump coronary artery bypass grafting.
gan: Antoniades, C, et al.
Cyhoeddwyd: (2009)

Altered plasma versus vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function, and inflammation.
gan: Antoniades, C, et al.
Cyhoeddwyd: (2007)

Novel role of 5-methyl-(6S)-tetrahydrofolate in mediating endothelial cell tetrahydrobiopterin in pregnancy and implications for gestational hypertension
gan: Dickinson, Y, et al.
Cyhoeddwyd: (2024)

Folic acid induces maximal vascular effects at dosage equivalent to the recommended daily allowance (RDA): Implications for folic acid treatment in fortified populations
gan: Antoniades, C, et al.
Cyhoeddwyd: (2007)

Mechanisms of increased vascular superoxide production in human diabetes mellitus: role of NAD(P)H oxidase and endothelial nitric oxide synthase.
gan: Guzik, T, et al.
Cyhoeddwyd: (2002)

MTHFR 677 C>T Polymorphism reveals functional importance for 5-methyltetrahydrofolate, not homocysteine, in regulation of vascular redox state and endothelial function in human atherosclerosis.
gan: Antoniades, C, et al.
Cyhoeddwyd: (2009)

UltraRapid communications : vascular superoxide production by NAD(P)H OxidaseAssociation with endothelial dysfunction and clinical risk factors
gan: Guzik, T, et al.
Cyhoeddwyd: (2000)

Effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease

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