Complement activation in patients with neuromyelitis optica.

Complement activation in patients with neuromyelitis optica.

عرض إصدارات أخرى (1)

The role of complement has been demonstrated in experimental models of neuromyelitis optica (NMO), however, only few studies have analysed complement components longitudinally in NMO patients. We measured serum or plasma concentrations of anti-C1q antibodies and complement split products C3a and C4a...

وصف كامل

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Nytrova, P, Potlukova, E, Kemlink, D, Woodhall, M, Horakova, D, Waters, P, Havrdova, E, Zivorova, D, Vincent, A, Trendelenburg, M
التنسيق:	Journal article
اللغة:	English
منشور في:	2014

مواد مشابهة

Complement activation in patients with neuromyelitis optica
حسب: Nytrova, P, وآخرون
منشور في: (2014)

C3a complement split product: possible biomarker of disease activity in patients with neuromyelitis optica
حسب: Nytrova, P, وآخرون
منشور في: (2013)

Intra-cerebral injection of neuromyelitis optica immunoglobulin G and human complement produces neuromyelitis optica lesions in mice.
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IgG subclasses of disease-specific antibodies and complement activation in-vitro in seronegative myasthenia gravis and neuromyelitis optica
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حسب: Tackley, G, وآخرون
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Myelin-oligodendrocyte glycoprotein antibody-mediated acute inflammatory demyelinating disease mimicking neuromyelitis optica
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Methotrexate is an alternative to azathioprine in neuromyelitis optica spectrum disorders with aquaporin-4 antibodies.
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IgG subclasses of disease-specific antibodies and complement activation in vitro in myasthenia gravis and neuromyelitis optica
حسب: Jacob, S, وآخرون
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Utility of aquaporin-4 antibody assay in patients with neuromyelitis optica spectrum disorders.
حسب: Kim, S, وآخرون
منشور في: (2013)

Antibodies to aquaporin-1 are not present in neuromyelitis optica
حسب: Waters, P, وآخرون
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Mechanisms of disease: aquaporin-4 antibodies in neuromyelitis optica.
حسب: Jarius, S, وآخرون
منشور في: (2008)

Complement activating antibodies to myelin oligodendrocyte glycoprotein in neuromyelitis optica and related disorders
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منشور في: (2011-12-01)

MYELIN-OLIGODENDROCYTE GLYCOPROTEIN ANTIBODY AS A CAUSE OF ACUTE DISSEMINATED ENCEPHALOMYELITIS WITH A NEUROMYELITIS OPTICA-LIKE PHENOTYPE IN ADULTS
حسب: Kitley, J, وآخرون
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Symptomatic, radiological and pathological involvement of the hypothalamus in neuromyelitis optica.
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Prognostic implications of aquaporin-4 antibody status in neuromyelitis optica patients.
حسب: Akman-Demir, G, وآخرون
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Detection of anti-aquaporin-4 antibodies in neuromyelitis optica: current status of the assays.
حسب: Waters, P, وآخرون
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Characterization of neuromyelitis optica and neuromyelitis optica spectrum disorder patients with a late onset.
حسب: Collongues, N, وآخرون
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Effect of IgG from seronegative and seropositive neuromyelitis optica patients on mouse brain
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Neuromyelitis optica spectrum disorders with aquaporin-4 and myelin-oligodendrocyte glycoprotein antibodies: a comparative study.
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Neuromyelitis optica IgG causes placental inflammation and fetal death.
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Rituximab abrogates aquaporin-4-specific germinal center activity in patients with neuromyelitis optica spectrum disorders
حسب: Damato, V, وآخرون
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