$Novel use of burosumab in refractory iron-induced FGF23-mediated hypophosphataemic osteomalacia$

Novel use of burosumab in refractory iron-induced FGF23-mediated hypophosphataemic osteomalacia

Bibliográfalaš dieđut
Váldodahkkit:	Amarnani, R, Travis, S, Javaid, MK
Materiálatiipa:	Journal article
Giella:	English
Almmustuhtton:	Oxford University Press 2020

Geahča maid

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Effects of burosumab on osteocalcin and bone mineral density in patient with 15-year history of nonremission tumor-induced osteomalacia initially treated with conventional therapy: Case report
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A case report to assess the safety and efficacy of Burosumab, an investigational antibody to FGF23, in a single pediatric patient with Epidermal Nevus Syndrome and associated hypophosphatemic rickets
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Combined treatment by burosumab and a calcimimetic can ameliorate hypophosphatemia due to excessive actions of FGF23 and PTH in adult XLH with tertiary hyperparathyroidism: A case report
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Predicting the impact of PHEX, FGF23 and DMP1 gene variants found in Malaysian Malay patients with Hypophosphataemic Rickets through in silico analysis of protein function and mRNA secondary structure
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Long-term safety in adults with X-linked Hypophosphatemia (XLH) treated with Burosumab, a fully human monoclonal antibody against FGF23: Final results of a phase 3 trial
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Genetic basis of hereditary hypophosphataemic rickets and phenotype presentation in children and adults
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Antibody-mediated activation of FGFR1 induces FGF23 production and hypophosphatemia.
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Prediction of the structural interface between fibroblast growth factor23 and Burosumab using alanine scanning and molecular docking
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