Anti-inflammatory effects of nicotinic acid in adipocytes demonstrated by suppression of fractalkine, RANTES, and MCP-1 and upregulation of adiponectin.
OBJECTIVE: A major site of action for the atheroprotective drug nicotinic acid (NA) is adipose tissue, via the G-protein-coupled receptor, GPR109A. Since, adipose tissue is an active secretory organ that contributes both positively and negatively to systemic inflammatory processes associated with c...
Glavni autori: | Digby, J, McNeill, E, Dyar, O, Lam, V, Greaves, D, Choudhury, R |
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Format: | Journal article |
Jezik: | English |
Izdano: |
2010
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Slični predmeti
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Anti-inflammatory effects of nicotinic acid in adipocytes demonstrated by suppression of fractalkine, RANTES, and MCP-1 and upregulation of adiponectin
od: Digby, J, i dr.
Izdano: (2010) -
Anti-inflammatory Effects of Nicotinic Acid: Suppression of Fractalkine, RANTES, and MCP-1 and Upregulation of Adiponectin in 3T3-L1 Adipocytes
od: Digby, J, i dr.
Izdano: (2008) -
Fractalkine promotes human monocyte survival via a reduction in oxidative stress.
od: White, G, i dr.
Izdano: (2014) -
Stimulation of Alpha7 Nicotinic Acetylcholine Receptor Attenuates Nicotine-Induced Upregulation of MMP, MCP-1, and RANTES through Modulating ERK1/2/AP-1 Signaling Pathway in RAW264.7 and MOVAS Cells
od: Liping Liu, i dr.
Izdano: (2017-01-01) -
Fractalkine: one chemokine, many functions.
od: White, G, i dr.
Izdano: (2009)