Evidence for relaxation to endothelium-derived hyperpolarizing factor (EDHF) in isolated small mesenteric arteries of the mouse

Evidence for relaxation to endothelium-derived hyperpolarizing factor (EDHF) in isolated small mesenteric arteries of the mouse

Podrobná bibliografie
Hlavní autoři:	Dora, K, Garland, C
Médium:	Conference item
Vydáno:	2001

Podobné jednotky

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Myoendothelial gap junctions may provide the pathway for EDHF in mouse mesenteric artery.
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K(Ca) channel blockers reveal hyperpolarization and relaxation to K+ in rat isolated mesenteric artery.
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Hyperpolarization and the endothelium
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Thromboxane receptor stimulation associated with loss of SKCa activity and reduced EDHF responses in the rat isolated mesenteric artery.
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Hyperpolarization and relaxation of vascular smooth muscle to endothelium-derived nitric oxide
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An indirect influence of phenylephrine on the release of endothelium-derived vasodilators in rat small mesenteric artery.
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EDH: Endothelium-dependent hyperpolarization and microvascular signaling.
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Endocannabinoids as endothelium-derived hyperpolarizing factors: The end of the road?
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Relaxation to authentic nitric oxide and SIN-1 in rat isolated mesenteric arteries: variable role for smooth muscle hyperpolarization.
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Possible role for K+ in endothelium-derived hyperpolarizing factor-linked dilatation in rat middle cerebral artery.
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K+ may act as an EDHF in rat middle cerebral artery
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Vascular hyperpolarization to β-adrenoceptor agonists evokes spreading dilatation in rat isolated mesenteric arteries.
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A crucial influence of precontraction on potassium-induced relaxation in the rat isolated mesenteric artery
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Spreading dilatation in rat mesenteric arteries associated with calcium-independent endothelial cell hyperpolarization.
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1-ethyl-2-benzimidazolinone activates endothelial cell IKCa and smooth muscle hyperpolarization in rat isolated mesenteric artery
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Heterocellular calcium signaling: A novel stimulus for the release of EDHF?
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Activation of endothelial cell IK(Ca) with 1-ethyl-2-benzimidazolinone evokes smooth muscle hyperpolarization in rat isolated mesenteric artery.
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Endothelium-Derived Hyperpolarizing Factor and Vascular Function
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A central role for endothelial cell potassium channels in EDHF-mediated responses
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NO and KATP channels underlie endotoxin-induced smooth muscle hyperpolarization in rat mesenteric resistance arteries.
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Reduced endothelium-dependent relaxation to anandamide in mesenteric arteries from young obese Zucker rats.
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A Role of Ion Channels in the Endothelium-Independent Relaxation of Rat Mesenteric Artery Induced by Resveratrol
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Hydrogen Peroxide Is an Endothelium-Derived Hyperpolarizing Factor in Animals and Humans
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Chronic Hypoxia Decreases Endothelial Connexin 40, Attenuates Endothelium‐Dependent Hyperpolarization–Mediated Relaxation in Small Distal Pulmonary Arteries, and Leads to Pulmonary Hypertension
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Vydáno: (2020-12-01)