Discordant response to exercise training in nnos knockout mice: Differential role of nNOS on exercise performance versus cardiac hypertrophy

Discordant response to exercise training in nnos knockout mice: Differential role of nNOS on exercise performance versus cardiac hypertrophy

Podrobná bibliografie
Hlavní autoři:	de Bono, J, Adlam, D, Zhang, M, Paterson, D, Casadei, B, Channon, K
Médium:	Conference item
Vydáno:	2007

Podobné jednotky

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Role of nNOS in the automatic control of cardiac excitability in cardiac physiological and pathophysiological states
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Cardiac cholinergic NO-cGMP signaling following acute myocardial infarction and nNOS gene transfer.
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Adenoviral nNOS gene transfer into the cardiac vagus increases protein expression within 9 hours and enhances vagal responsiveness in the pig
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BH4 increases nNOS Activity and preserves left ventricular function in diabetes
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A role for nNOS in mediating stress and female sexual behavior in mice
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Effect of in vivo gene transfer of nNOS in the PVN on renal nerve discharge in rats.
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The PSD-95/nNOS complex: New drugs for depression?
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Presence of atherosclerosis determines the vascular but not the cardiac effects of exercise training through endothelial NOS activity
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Neuronal nitric oxide synthase-rescue of dystrophin/utrophin double knockout mice does not require nNOS localization to the cell membrane.
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Neuronal NOS (nNOS) is expressed in human osteocytes: Multiple pathways of NO regulation?
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Mechanisms of NOS1AP action on NMDA receptor-nNOS signalling
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Are myocardial eNOS and nNOS involved in the beta-adrenergic and muscarinic regulation of inotropy? A systematic investigation.
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nNOS gene transfer to RVLM improves baroreflex function in rats with chronic heart failure.
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nNOS V eNOS mediated regulation of beta adrenergic inotropic and lusitropic responses in left ventricular myocytes
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Phenotyping of nNOS neurons in the postnatal and adult female mouse hypothalamus
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Sodium channel and nNOS expression following inferior alveolar nerve
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nNOS expression following inferior alveolar nerve injury in the ferret.
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Novel long-range inhibitory nNOS-expressing hippocampal cells
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nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction
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nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction.
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