DNA AND RNA STUDIES INTO THE NPM-ALK REARRANGEMENT IN KI-1 POSITIVE ANAPLASTIC LARGE-CELL LYMPHOMA, DIAGNOSTIC APPLICATIONS

DNA AND RNA STUDIES INTO THE NPM-ALK REARRANGEMENT IN KI-1 POSITIVE ANAPLASTIC LARGE-CELL LYMPHOMA, DIAGNOSTIC APPLICATIONS

Xehetasun bibliografikoak
Egile Nagusiak:	Waggott, W, Lo, Y, Bastard, C, Gatter, K, Leroux, D, Mason, D, Boultwood, J, Wainscoat, J
Formatua:	Journal article
Argitaratua:	1994

Antzeko izenburuak

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The tyrosine phosphatase Shp2 interacts with NPM-ALK and regulates anaplastic lymphoma cell growth and migration.
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Correction: Dual inhibition of IGF-IR and ALK as an effective strategy to eradicate NPM-ALK+ T-cell lymphoma
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Quantification of minimal disseminated disease by quantitative polymerase chain reaction and digital polymerase chain reaction for NPM-ALK as a prognostic factor in children with anaplastic large cell lymphoma
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<i>DUSP22</i> rearrangement is associated with a distinctive immunophenotype but not outcome in patients with systemic ALK-negative anaplastic large cell lymphoma
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Acquired L1196M ALK mutation in anaplastic lymphoma kinase‐positive anaplastic large cell lymphoma during alectinib administration
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<i>DUSP22</i>-rearranged ALK-negative anaplastic large cell lymphoma is a pathogenetically distinct disease but can have variable clinical outcome
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Alectinib, an anaplastic lymphoma kinase (ALK) inhibitor, as a bridge to allogeneic stem cell transplantation in a patient with ALK‐positive anaplastic large‐cell lymphoma refractory to chemotherapy and brentuximab vedotin
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NIPA (Nuclear Interaction Partner of ALK) Is Crucial for Effective NPM-ALK Mediated Lymphomagenesis
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