Failure to block adhesion of Plasmodium falciparum-infected erythrocytes to ICAM-1 with soluble ICAM-1.
The adhesion of Plasmodium falciparum-infected erythrocytes is thought to play a central role in the pathogenesis of severe malaria. ICAM-1 has been identified as one of the host receptors for parasitized erythrocytes and has been implicated as being involved in progression to cerebral malaria. Thus...
المؤلفون الرئيسيون: | Craig, A, Pinches, R, Khan, S, Roberts, D, Turner, G, Newbold, C, Berendt, A |
---|---|
التنسيق: | Journal article |
اللغة: | English |
منشور في: |
1997
|
مواد مشابهة
-
INTERACTIONS OF MALARIA-INFECTED ERYTHROCYTES WITH ICAM-1 AND VASCULAR ENDOTHELIUM
حسب: Berendt, A, وآخرون
منشور في: (1993) -
The binding site on ICAM-1 for Plasmodium falciparum-infected erythrocytes overlaps, but is distinct from, the LFA-1-binding site.
حسب: Berendt, A, وآخرون
منشور في: (1992) -
An analysis of ICAM-1 in Plasmodium falciparum malaria.
حسب: Ochola, L, وآخرون
منشور في: (2008) -
Structural insights into diverse modes of ICAM-1 binding by Plasmodium falciparum-infected erythrocytes
حسب: Lennartz, F, وآخرون
منشور في: (2019) -
Rolling and stationary cytoadhesion of red blood cells parasitized by Plasmodium falciparum: separate roles for ICAM-1, CD36 and thrombospondin.
حسب: Cooke, B, وآخرون
منشور في: (1994)