Five quantitative trait loci control radiation-induced adenoma multiplicity in Mom 1R ApcMin/+ mice
Ionising radiation is a carcinogen capable of inducing tumours, including colorectal cancer, in both humans and animals. By backcrossing a recombinant line of ApcMin/+ mice to the inbred BALB/c mouse strain, which is unusually sensitive to radiation-induced tumour development, we obtained panels of...
Main Authors: | Elahi, E, Suraweera, N, Volikos, E, Haines, J, Brown, N, Davidson, G, Churchman, M, Ilyas, M, Tomlinson, I, Silver, A |
---|---|
格式: | Journal article |
语言: | English |
出版: |
Public Library of Science
2009
|
相似书籍
-
Five quantitative trait loci control radiation-induced adenoma multiplicity in Mom1R Apc Min/+ mice.
由: Elahi, E, et al.
出版: (2009) -
Five quantitative trait loci control radiation-induced adenoma multiplicity in Mom1R Apc Min/+ mice.
由: Eiram Elahi, et al.
出版: (2009-01-01) -
Genetic basis of variation in adenoma multiplicity in ApcMin/+ Mom1S mice.
由: Haines, J, et al.
出版: (2005) -
Myh deficiency enhances intestinal tumorigenesis in multiple intestinal neoplasia (ApcMin/+) mice.
由: Sieber, O, et al.
出版: (2004) -
Platelet status in cancer cachexia progression in ApcMin/+ mice
由: Patrice Cunningham, et al.
出版: (2023-08-01)