The role of rank ligand in RA synovial macrophage-osteoclast differentiation

The role of rank ligand in RA synovial macrophage-osteoclast differentiation

Sonraí bibleagrafaíochta
Príomhchruthaitheoirí:	Itonaga, I, Fujikawa, Y, Sabokbar, A, Murray, D, Athanasou, N
Formáid:	Journal article
Foilsithe / Cruthaithe:	2000

Míreanna comhchosúla

Rheumatoid arthritis synovial macrophage-osteoclast differentiation is osteoprotegerin ligand-dependent.
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Synovial macrophage-osteoclast differentiation in inflammatory arthritis.
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Effect of osteoprotegerin and osteoprotegerin ligand on osteoclast formation by arthroplasty membrane derived macrophages.
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Synovial fluid macrophages are capable of osteoclast differentiation.
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Human osteoclast formation and bone resorption by monocytes and synovial macrophages in rheumatoid arthritis.
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Human osteoclast formation and bone resorption by a mechanism independent of RANK/RANKL: Role of IL-6 and IL-11.
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Human arthroplasty derived macrophages differentiate into osteoclastic bone resorbing cells.
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Stimulation of osteoclast formation by inflammatory synovial fluid.
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Stimulation of osteoclast formation by inflammatory synovial fluid
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Synovial fluid macrophages are capable of osteoclast formation and resorption.
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Synovial fluid promotes macrophage and osteoclast formation in inflammatory arthritis
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Increased osteoclastic differentiation by PMMA particle-associated macrophages. Inhibitory effect by interleukin 4 and leukemia inhibitory factor.
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The effect of macrophage-colony stimulating factor and other humoral factors (interleukin-1, -3, -6, and -11, tumor necrosis factor-alpha, and granulocyte macrophage-colony stimulating factor) on human osteoclast formation from circulating cells.
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Rheumatoid and crystal arthritic synovial fibroblasts induce osteoclast formation
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Major sex and age differences in human osteoclast formation from circulating precursors: Implications for the pathogenesis of primary osteoporosis
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Macrophage-osteoclast differentiation and bone resorption in osteoarthrotic subchondral acetabular cysts.
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Proinflammatory cytokine (TNFalpha/IL-1alpha) induction of human osteoclast formation.
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Humoral factors act directly on circulating osteoclast precursors to control osteoclast formation
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RESIDENT AND INFLAMMATORY TISSUE MACROPHAGES DIFFERENTIATE INTO OSTEOCLASTIC BONE-RESORBING CELLS
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Human tumour associated macrophages are capable of osteoclastic differentiation and bone resorption
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Interleukin-6 and interleukin-11 support human osteoclast formation by a RANKL-independent mechanism.
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Human macrophage-osteoclast differentiation and pathological bone resorption in inflammation and malignancy
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Isolation of human osteoclasts formed in vitro: hormonal effects on the bone-resorbing activity of human osteoclasts.
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Sarcoma-associated macrophages differentiate into osteoclastic bone-resorbing cells
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Human mesenchymal tumour-associated macrophages differentiate into osteoclastic bone-resorbing cells.
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Transforming growth factor-beta induces osteoclast formation in the absence of RANKL.
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Human osteoclasts differentiate from a subpopulation of circulating monocytes.
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RANKL and M-CSF are required for osteoclast differentiation by macrophages in breast cancer
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1,25-Dihydroxyvitamin D(3) and prostaglandin E(2) act directly on circulating human osteoclast precursors.
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Cellular and humoral mechanisms of osteoclast formation in Gorham-Stout disease (GSD)
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RANKL-dependent and RANKL-independent mechanisms of macrophage-osteoclast differentiation in breast cancer.
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Bone stromal cells in pagetic bone and Paget's sarcoma express RANKL and support human osteoclast formation.
de réir: Sun, S, et al.
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Cellular and humoral mechanisms of osteoclast formation and bone resorption in Gorham-Stout disease.
de réir: Hirayama, T, et al.
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Arthroplasty membrane-derived fibroblasts directly induce osteoclast formation and osteolysis in aseptic loosening.
de réir: Sabokbar, A, et al.
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Human osteoblasts support human monocyte-osteoclast differentiation
de réir: Neale, S, et al.
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Rankl-independent induction of human osteoclast formation and bone resorption by TGF-beta
de réir: Itonaga, I, et al.
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The human osteoclast precursor circulates in the monocyte fraction.
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IL-6 induces human osteoclast formation and bone resorption independent of RANK/RANKL interaction
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