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The Prolyl Hydroxylase Inhibitor BIQ/FG-2216 Stabilises HIF-1 alpha and Upregulates VEGF in vitro: A Potential Strategy For Difficult CTO Lesions in Patients.

The Prolyl Hydroxylase Inhibitor BIQ/FG-2216 Stabilises HIF-1 alpha and Upregulates VEGF in vitro: A Potential Strategy For Difficult CTO Lesions in Patients.

Manylion Llyfryddiaeth
Prif Awduron: Barnett, G, Flashman, E, Kelly, D, Schofield, C, Gershlick, A
Fformat: Conference item
Cyhoeddwyd: 2013
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Eitemau Tebyg

  • 74 Pro-Angiogenic Effects of the Prolyl-hydroxylase Inhibitor FG-2216/BIQ: Therapeutic Angiogenesis for the Treatment of Chronic Total Occlusions.
    gan: Barnett, G, et al.
    Cyhoeddwyd: (2014)
  • Pro-angiogenic effects of proly-hydroxylase inhibitor FG-2216 and potential for use in a novel strategy of therapeutic angiogenesis for difficult cases of coronary total occlusion
    gan: Barnett, G, et al.
    Cyhoeddwyd: (2014)
  • Enhanced Endothelial Tubule Formation in Response to Proly-Hydroxylase Inhibitor FG-2216: A Step Towards Therapeutic Angiogenesis in Chronic Total Occlusions
    gan: Barnett, G, et al.
    Cyhoeddwyd: (2012)
  • Investigating the dependence of the hypoxia-inducible factor hydroxylases (factor inhibiting HIF and prolyl hydroxylase domain 2) on ascorbate and other reducing agents.
    gan: Flashman, E, et al.
    Cyhoeddwyd: (2010)
  • Regulation of HIF: prolyl hydroxylases.
    gan: Stolze, I, et al.
    Cyhoeddwyd: (2006)

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