Tetrahydrobiopterin: coupling nitric oxide and redox signalling in cardiovascular disease

Tetrahydrobiopterin: coupling nitric oxide and redox signalling in cardiovascular disease

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Channon, K
التنسيق:	Conference item
منشور في:	2012

مواد مشابهة

Tetrahydrobiopterin and nitric oxide synthase recouplers
حسب: Channon, KM
منشور في: (2020)

Tetrahydrobiopterin: regulator of endothelial nitric oxide synthase in vascular disease.
حسب: Channon, K
منشور في: (2004)

Regulation of endothelial nitric oxide synthase by tetrahydrobiopterin in vascular disease.
حسب: Alp, N, وآخرون
منشور في: (2004)

Integrated redox sensor and effector functions for tetrahydrobiopterin- and glutathionylation-dependent endothelial nitric-oxide synthase uncoupling.
حسب: Crabtree, M, وآخرون
منشور في: (2013)

Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin-mediated endothelial nitric oxide synthase coupling.
حسب: Antoniades, C, وآخرون
منشور في: (2011)

Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin- mediated endothelial nitric oxide synthase coupling
حسب: Antoniades, C, وآخرون
منشور في: (2011)

Tetrahydrobiopterin protects against hypertrophic heart disease independent of myocardial nitric oxide synthase coupling.
حسب: Hashimoto, T, وآخرون
منشور في: (2016)

Tetrahydrobiopterin protects against hypertrophic heart disease independent of myocardial nitric oxide synthase coupling
حسب: Hashimoto, T, وآخرون
منشور في: (2016)

Regulation of endothelial nitric oxide synthase (eNOS) by tetrahydrobiopterin in vascular disease
حسب: Alp, N, وآخرون
منشور في: (2004)

Statin treatment improves vascular redox state through a tetrahydrobiopterin-mediated re-coupling of endothelial nitric oxide synthase, in human atherosclerosis
حسب: Antoniades, C, وآخرون
منشور في: (2011)

RAPID, DIRECT EFFECTS OF STATIN TREATMENT ON ARTERIAL REDOX STATE AND NITRIC OXIDE BIOAVAILABILITY IN HUMAN ATHEROSCLEROSIS VIA TETRAHYDROBIOPTERIN-MEDIATED ENOS COUPLING
حسب: Antoniades, C, وآخرون
منشور في: (2011)

The role of tetrahydrobiopterin in inflammation and cardiovascular disease.
حسب: McNeill, E, وآخرون
منشور في: (2012)

Tetrahydrobiopterin in cardiovascular health and disease.
حسب: Bendall, J, وآخرون
منشور في: (2014)

Tetrahydrobiopterin deficiency uncouples nitric oxide synthase and causes pulmonary hypertension
حسب: Khoo, J, وآخرون
منشور في: (2004)

Tetrahydrobiopterin: a vascular redox target to improve endothelial function.
حسب: Channon, K
منشور في: (2012)

Dihydrofolate reductase protects endothelial nitric oxide synthase from uncoupling in tetrahydrobiopterin deficiency.
حسب: Crabtree, M, وآخرون
منشور في: (2011)

Dihydrofolate reductase protects endothelial nitric oxide synthase from uncoupling in tetrahydrobiopterin deficiency
حسب: Crabtree, M, وآخرون
منشور في: (2011)

Endothelial tetrahydrobiopterin deficiency accelerates atherosclerotic progression by nitric oxide synthase uncoupling
حسب: Khoo, J, وآخرون
منشور في: (2005)

Free radicals and redox signalling in cardiovascular disease.
حسب: Shah, A, وآخرون
منشور في: (2004)

Tetrahydrobiopterin Prevents Lung Ischemia/reperfusion-Induced Uncoupling of Endothelial Nitric Oxide Synthase
حسب: Juni, R, وآخرون
منشور في: (2012)

Tetrahydrobiopterin: Pleiotropic roles in cardiovascular pathophysiology
حسب: Cunnington, C, وآخرون
منشور في: (2010)

Tetrahydrobiopterin: pleiotropic roles in cardiovascular pathophysiology.
حسب: Cunnington, C, وآخرون
منشور في: (2010)

Endothelial nitric oxide synthase dysfunction in diabetic mice: importance of tetrahydrobiopterin in eNOS dimerisation.
حسب: Cai, S, وآخرون
منشور في: (2005)

Redox sensor and effector functions of tetrahydrobiopterin and the mitochondria in molecular models of cardiovascular disease
حسب: Bailey, J
منشور في: (2018)

Free radicals and redox signalling in cardiovascular disease: introduction
حسب: Shah, A, وآخرون
منشور في: (2004)

Reduced levels of tetrahydrobiopterin in diabetic mouse model: Effects on endothelial function and nitric oxide (NO) synthase dimerisation
حسب: Cai, S, وآخرون
منشور في: (2004)

Recoupling the cardiac nitric oxide synthases: tetrahydrobiopterin synthesis and recycling.
حسب: Alkaitis, MS, وآخرون
منشور في: (2012)

Recoupling the cardiac nitric oxide synthases: Tetrahydrobiopterin synthesis and recycling
حسب: Alkaitis, MS, وآخرون
منشور في: (2012)

Nitric oxide Bioavailability is preserved in diabetes by increased endothelial tetrahydrobiopterin synthesis in targeted GTP cyclohydrolase transgenic mice
حسب: Alp, N, وآخرون
منشور في: (2003)

Quantitative regulation of intracellular endothelial nitric-oxide synthase (eNOS) coupling by both tetrahydrobiopterin-eNOS stoichiometry and biopterin redox status: insights from cells with tet-regulated GTP cyclohydrolase I expression.
حسب: Crabtree, M, وآخرون
منشور في: (2009)

A Novel Role for Endothelial Tetrahydrobiopterin in Mitochondrial Redox Balance
حسب: Bailey, J, وآخرون
منشور في: (2017)

Critical role for tetrahydrobiopterin recycling by dihydrofolate reductase in regulation of endothelial nitric-oxide synthase coupling: relative importance of the de novo biopterin synthesis versus salvage pathways.
حسب: Crabtree, M, وآخرون
منشور في: (2009)

Prevention of hypoxia-induced pulmonary hypertension and vascular remodelling by targeting endothelial tetrahydrobiopterin-dependent nitric oxide synthase regulation
حسب: Khoo, J, وآخرون
منشور في: (2005)

5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
حسب: Antoniades, C, وآخرون
منشور في: (2006)

Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling
حسب: Antoniades, C, وآخرون
منشور في: (2008)

Chronic treatment with folic acid improves nitric oxide bioavailability and decreases superoxide production in human vessels, through tetrahydrobiopterin-mediated improvement of eNOS coupling
حسب: Antoniades, C, وآخرون
منشور في: (2007)

P683The role of nitric oxide synthase (NOS) and its essential cofactor tetrahydrobiopterin (BH4) in diabetic cardiomyopathy.
حسب: Duglan, D, وآخرون
منشور في: (2014)

Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
حسب: Alp, N, وآخرون
منشور في: (2003)

GFRP tonically inhibits production of tetrahydrobiopterin and nitric oxide in marine endothelial cells
حسب: Kolinsky, M, وآخرون
منشور في: (2004)

Adenovirus-mediated GTP cyclohydrolase I gene delivery augments tetrahydrobiopterin and restores nitric oxide synthase function in hyperglycaemic human aortic endothelial cells
حسب: Cai, S, وآخرون
منشور في: (2004)