XIAP Restricts TNF- and RIP3-Dependent Cell Death and Inflammasome Activation

X-linked inhibitor of apoptosis protein (XIAP) has been identified as a potent regulator of innate immune responses, and loss-of-function mutations inXIAP cause the development of the X-linked lymphoproliferative syndrome type 2 (XLP-2) in humans. Using gene-targeted mice, we show that loss of XIAPo...

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Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Yabal, M, Müller, N, Adler, H, Knies, N, Groß, C, Damgaard, R, Kanegane, H, Ringelhan, M, Kaufmann, T, Heikenwälder, M, Strasser, A, Groß, O, Ruland, J, Peschel, C, Gyrd-Hansen, M, Jost, P
Μορφή:	Journal article
Γλώσσα:	English
Έκδοση:	Elsevier 2014

XIAP restricts TNF- and RIP3-dependent cell death and inflammasome activation. ανά Yabal, M, Müller, N, Adler, H, Knies, N, Groß, C, Damgaard, R, Kanegane, H, Ringelhan, M, Kaufmann, T, Heikenwälder, M, Strasser, A, Groß, O, Ruland, J, Peschel, C, Gyrd-Hansen, M, Jost, P

Έκδοση 2014

Journal article

XIAP Restricts TNF- and RIP3-Dependent Cell Death and Inflammasome Activation

XIAP restricts TNF- and RIP3-dependent cell death and inflammasome activation. ανά Yabal, M, Müller, N, Adler, H, Knies, N, Groß, C, Damgaard, R, Kanegane, H, Ringelhan, M, Kaufmann, T, Heikenwälder, M, Strasser, A, Groß, O, Ruland, J, Peschel, C, Gyrd-Hansen, M, Jost, P

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