XIAP Restricts TNF- and RIP3-Dependent Cell Death and Inflammasome Activation

X-linked inhibitor of apoptosis protein (XIAP) has been identified as a potent regulator of innate immune responses, and loss-of-function mutations inXIAP cause the development of the X-linked lymphoproliferative syndrome type 2 (XLP-2) in humans. Using gene-targeted mice, we show that loss of XIAPo...

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Những tác giả chính:	Yabal, M, Müller, N, Adler, H, Knies, N, Groß, C, Damgaard, R, Kanegane, H, Ringelhan, M, Kaufmann, T, Heikenwälder, M, Strasser, A, Groß, O, Ruland, J, Peschel, C, Gyrd-Hansen, M, Jost, P
Định dạng:	Journal article
Ngôn ngữ:	English
Được phát hành:	Elsevier 2014

XIAP restricts TNF- and RIP3-dependent cell death and inflammasome activation. Bằng Yabal, M, Müller, N, Adler, H, Knies, N, Groß, C, Damgaard, R, Kanegane, H, Ringelhan, M, Kaufmann, T, Heikenwälder, M, Strasser, A, Groß, O, Ruland, J, Peschel, C, Gyrd-Hansen, M, Jost, P

Được phát hành 2014

Journal article

XIAP Restricts TNF- and RIP3-Dependent Cell Death and Inflammasome Activation

XIAP restricts TNF- and RIP3-dependent cell death and inflammasome activation. Bằng Yabal, M, Müller, N, Adler, H, Knies, N, Groß, C, Damgaard, R, Kanegane, H, Ringelhan, M, Kaufmann, T, Heikenwälder, M, Strasser, A, Groß, O, Ruland, J, Peschel, C, Gyrd-Hansen, M, Jost, P

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