Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase
Atrial fibrillation (AF) is a growing public health burden, and its treatment remains a challenge. AF leads to electrical remodeling of the atria, which in turn promotes AF maintenance and resistance to treatment. Although remodeling has long been a therapeutic target in AF, its causes remain poorly...
Main Authors: | Reilly, S, Liu, X, Carnicer, R, Recalde, A, Muszkiewicz, A, Jayaram, R, Carena, M, Wijesurendra, R, Stefanini, M, Surdo, N, Lomas, O, Ratnatunga, C, Sayeed, R, Krasopoulos, G, Rajakumar, T, Bueno-Orovio, A, Verheule, S, Fulga, T, Rodriguez, B, Schotten, U, Casadei, B |
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Format: | Journal article |
Published: |
American Association for the Advancement of Science
2016
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