Increased renal C5a receptor expression and systemic complement activation in brain dead donors

Increased renal C5a receptor expression and systemic complement activation in brain dead donors

Podrobná bibliografie
Hlavní autoři:	van Werkhoven, M, Damman, J, Daha, MR, Leuvenink, H, Ploeg, R, van Goor, H, Hillebrands, J, van Son, W, Seelen, MAJ
Médium:	Conference item
Vydáno:	2011

Podobné jednotky

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SYSTEMIC AND LOCAL RENAL COMPLEMENT ACTIVATION IN BRAIN-DEAD DONORS
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Crosstalk between Complement and Toll-like Receptor Activation in Relation to Donor Brain Death and Renal Ischemia-Reperfusion Injury (vol 11, pg 660, 2011)
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Erratum: Crosstalk between complement and toll-like receptor activation in relation to donor brain death and renal ischemia-reperfusion injury (American Journal of Transplantation (2011) 11 (660-669) DOI: 10.1111/j.1600-6143.2011. 03475.x)
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Targeting complement activation in brain-dead donors improves renal function after transplantation.
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Systemic complement activation in deceased donors is associated with acute rejection after renal transplantation in the recipient.
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Local renal complement C3 induction by donor brain death is associated with reduced renal allograft function after transplantation.
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Local renal upregulation of complement C3 and fibrinogen as part of the acute phase response in brain-dead donors
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Lectin complement pathway gene profile of the donor and recipient does not influence graft outcome after kidney transplantation
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Lectin complement pathway gene profile of the donor and recipient does not influence graft outcome after kidney transplantation.
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Increased intestinal permeability in the brain dead donor
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Toll-Like Receptor Family Polymorphisms Are Associated with Primary Renal Diseases but Not with Renal Outcomes Following Kidney Transplantation.
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Donor pretreatment with carbamylated erythropoietin in a brain death model reduces inflammation more effectively than erythropoietin while preserving renal function.
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Proteomic analysis of machine perfusion solution from brain dead donor kidneys reveals that elevated complement, cytoskeleton and lipid metabolism proteins are associated with 1-year outcome.
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ADAM17 up-regulation in renal transplant dysfunction and non-transplant-related renal fibrosis.
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Inflammation and structural changes in donor intestine and liver after brain death induction.
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ARA290 Protects Against Renal Ischemia/Reperfusion Injury in Pigs
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Brain death causes structural and inflammatory changes in donor intestine.
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