CD4 T cells from malaria-nonexposed individuals respond to the CD36-Binding Domain of Plasmodium falciparum erythrocyte membrane protein-1 via an MHC class II-TCR-independent pathway.

مواد مشابهة

• CD4 T cell responses to a variant antigen of the malaria parasite Plasmodium falciparum, erythrocyte membrane protein-1, in individuals living in malaria-endemic areas.
  حسب: Allsopp, C, وآخرون
  منشور في: (2002)
• Sulfated glycoconjugates enhance CD36-dependent adhesion of Plasmodium falciparum-infected erythrocytes to human microvascular endothelial cells.
  حسب: McCormick, C, وآخرون
  منشور في: (2000)
• Sickle-trait hemoglobin reduces adhesion to both CD36 and EPCR by Plasmodium falciparum-infected erythrocytes.
  حسب: Jens E V Petersen, وآخرون
  منشور في: (2021-06-01)
• Cellular responses to <it>Plasmodium falciparum</it> erythrocyte membrane protein-1: use of relatively conserved synthetic peptide pools to determine CD4 T cell responses in malaria-exposed individuals in Benin, West Africa
  حسب: Sanni Ambaliou, وآخرون
  منشور في: (2002-04-01)
• Intercellular adhesion molecule-1 and CD36 synergize to mediate adherence of Plasmodium falciparum-infected erythrocytes to cultured human microvascular endothelial cells.
  حسب: McCormick, C, وآخرون
  منشور في: (1997)