A cardiac nitric oxide synthase 1 regulates contraction and calcium fluxes - a novel mechanism in the autocrine control of cardiac function

A cardiac nitric oxide synthase 1 regulates contraction and calcium fluxes - a novel mechanism in the autocrine control of cardiac function

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Ashley, E, Sears, C, Lygate, C, Bryant, S, Wallis, H, Neubauer, S, Casadei, B
التنسيق:	Conference item
منشور في:	2002

مواد مشابهة

Cardiac neuronal nitric oxide synthase isoform regulates myocardial contraction and calcium handling.
حسب: Sears, C, وآخرون
منشور في: (2003)

A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
حسب: Sears, C, وآخرون
منشور في: (2002)

A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
حسب: Sears, C, وآخرون
منشور في: (2002)

Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left-ventricular contraction and relaxation in vivo
حسب: Ashley, E, وآخرون
منشور في: (2003)

Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left ventricular contraction and relaxation in vivo
حسب: Ashley, E, وآخرون
منشور في: (2003)

Nitric oxide control of cardiac function: is neuronal nitric oxide synthase a key component?
حسب: Sears, C, وآخرون
منشور في: (2004)

Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
حسب: Ashley, E, وآخرون
منشور في: (2002)

Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
حسب: Zhang, Y, وآخرون
منشور في: (2003)

Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
حسب: Sears, C, وآخرون
منشور في: (2001)

Nitric-oxide-mediated regulation of cardiac contractility and stretch responses.
حسب: Casadei, B, وآخرون
منشور في: (2003)

Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
حسب: Ashley, E, وآخرون
منشور في: (2001)

Effect of disruption of the nNOS gene on myocardial calcium current and contraction
حسب: Sears, C, وآخرون
منشور في: (2001)

The role of nitric oxide in the autonomic control of cardiac function
حسب: Sears, C
منشور في: (1998)

Selective gene deletion of the neuronal nitric oxide synthase (nNOS) accelerates pathological left ventricular remodelling and functional deterioration after myocardial infarction (MI)
حسب: Dawson, D, وآخرون
منشور في: (2005)

Recoupling the cardiac nitric oxide synthases: tetrahydrobiopterin synthesis and recycling.
حسب: Alkaitis, MS, وآخرون
منشور في: (2012)

Recoupling the cardiac nitric oxide synthases: Tetrahydrobiopterin synthesis and recycling
حسب: Alkaitis, MS, وآخرون
منشور في: (2012)

Does nitric oxide modulate cardiac ryanodine receptor function? Implications for excitation-contraction coupling.
حسب: Lim, G, وآخرون
منشور في: (2008)

DISTINCTIVE ROLE FOR PROTEIN PHOSPHATASES AND PROTEIN KINASES IN THE REGULATION OF MYOCARDIAL CONTRACTION BY THE NEURONAL NITRIC OXIDE SYNTHASE
حسب: Zhang, Y, وآخرون
منشور في: (2010)

Neuronal nitric oxide synthase-CAPON regulation of cardiac sympathetic activity in the development of hypertension
حسب: Lu, C
منشور في: (2015)

Nitric oxide-mediated regulation of cardiac contractility and stretch responses
حسب: Casadei, B
منشور في: (2004)

200 Nitric oxide promotes insulin-independent glucose uptake and preserves cardiac function and energetics in diabetes
حسب: Ziberna, K, وآخرون
منشور في: (2017)

NEURONAL NITRIC OXIDE SYNTHASE GENE DISRUPTION REDUCES THE DIASTOLIC CALCIUM LEAK FROM THE RYANODINE RECEPTOR: IMPLICATIONS FOR EXCITATION-CONTRACTION COUPLING IN HEART FAILURE
حسب: Lim, G, وآخرون
منشور في: (2009)

Nitric oxide synthases in heart failure.
حسب: Carnicer, R, وآخرون
منشور في: (2013)

Protective vascular and cardiac effects of inducible nitric oxide synthase in mice with hyperhomocysteinemia.
حسب: Sanjana Dayal, وآخرون
منشور في: (2014-01-01)

The subcellular localisation of neuronal nitric oxide synthase determines the downstream effects of NO on myocardial function
حسب: Carnicer, R, وآخرون
منشور في: (2017)

Neuronal nitric oxide synthase gene transfer promotes cardiac vagal gain of function.
حسب: Mohan, R, وآخرون
منشور في: (2002)

Targeting neuronal nitric oxide synthase with gene transfer to modulate cardiac autonomic function.
حسب: Mohan, R, وآخرون
منشور في: (2004)

Targeting cardiac sympatho-vagal imbalance using gene transfer of nitric oxide synthase.
حسب: Danson, E, وآخرون
منشور في: (2009)

Autocrine and paracrine nitric oxide regulate attachment of human osteoclasts.
حسب: Yaroslavskiy, B, وآخرون
منشور في: (2004)

MODELLING CALCIUM HANDLING MECHANISMS IN NORMAL AND NEURONAL NITRIC OXIDE SYNTHASE KNOCK-OUT MOUSE HEARTS
حسب: Li, L, وآخرون
منشور في: (2009)

The emerging role of neuronal nitric oxide synthase in the regulation of myocardial function.
حسب: Casadei, B
منشور في: (2006)

Neuronal nitric oxide synthase gene transfer decreases [Ca2+]i in cardiac sympathetic neurons.
حسب: Wang, L, وآخرون
منشور في: (2007)

Letter by Antoniades et al regarding article, "Uncoupled cardiac nitric oxide synthase mediates diastolic dysfunction".
حسب: Antoniades, C, وآخرون
منشور في: (2010)

Neuronal nitric oxide synthase and human vascular regulation.
حسب: Melikian, N, وآخرون
منشور في: (2009)

Response of cardiac endothelial nitric oxide synthase to plasma viscosity modulation in acute isovolemic hemodilution
حسب: Kanyanatt Kanokwiroon, وآخرون
منشور في: (2014-01-01)

Differential role of endothelial versus neuronal nitric oxide synthase in the regulation of coronary blood flow during pacing-induced increases in cardiac workload.
حسب: Shabeeh, H, وآخرون
منشور في: (2013)

Inducible nitric oxide synthase-derived extracellular nitric oxide flux regulates proinflammatory responses at the single cell level
حسب: Veena Somasundaram, وآخرون
منشور في: (2020-01-01)

Investigating cardiac energetics in heart failure.
حسب: Lygate, C, وآخرون
منشور في: (2013)

Investigating cardiac energetics in heart failure
حسب: Lygate, C, وآخرون
منشور في: (2013)

A cardiac-null mutation of Prdm16 causes hypotension in mice with cardiac hypertrophy via increased nitric oxide synthase 1.
حسب: Ji-One Kang, وآخرون
منشور في: (2022-01-01)