A cardiac nitric oxide synthase 1 regulates contraction and calcium fluxes - a novel mechanism in the autocrine control of cardiac function

A cardiac nitric oxide synthase 1 regulates contraction and calcium fluxes - a novel mechanism in the autocrine control of cardiac function

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Ashley, E, Sears, C, Lygate, C, Bryant, S, Wallis, H, Neubauer, S, Casadei, B
Μορφή:	Conference item
Έκδοση:	2002

Παρόμοια τεκμήρια

Cardiac neuronal nitric oxide synthase isoform regulates myocardial contraction and calcium handling.
ανά: Sears, C, κ.ά.
Έκδοση: (2003)

A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
ανά: Sears, C, κ.ά.
Έκδοση: (2002)

A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
ανά: Sears, C, κ.ά.
Έκδοση: (2002)

Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left-ventricular contraction and relaxation in vivo
ανά: Ashley, E, κ.ά.
Έκδοση: (2003)

Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left ventricular contraction and relaxation in vivo
ανά: Ashley, E, κ.ά.
Έκδοση: (2003)

Nitric oxide control of cardiac function: is neuronal nitric oxide synthase a key component?
ανά: Sears, C, κ.ά.
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Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
ανά: Ashley, E, κ.ά.
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Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
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Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
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Nitric-oxide-mediated regulation of cardiac contractility and stretch responses.
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Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
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Effect of disruption of the nNOS gene on myocardial calcium current and contraction
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The role of nitric oxide in the autonomic control of cardiac function
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Selective gene deletion of the neuronal nitric oxide synthase (nNOS) accelerates pathological left ventricular remodelling and functional deterioration after myocardial infarction (MI)
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Recoupling the cardiac nitric oxide synthases: tetrahydrobiopterin synthesis and recycling.
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Recoupling the cardiac nitric oxide synthases: Tetrahydrobiopterin synthesis and recycling
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Does nitric oxide modulate cardiac ryanodine receptor function? Implications for excitation-contraction coupling.
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DISTINCTIVE ROLE FOR PROTEIN PHOSPHATASES AND PROTEIN KINASES IN THE REGULATION OF MYOCARDIAL CONTRACTION BY THE NEURONAL NITRIC OXIDE SYNTHASE
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Neuronal nitric oxide synthase-CAPON regulation of cardiac sympathetic activity in the development of hypertension
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Nitric oxide-mediated regulation of cardiac contractility and stretch responses
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200 Nitric oxide promotes insulin-independent glucose uptake and preserves cardiac function and energetics in diabetes
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NEURONAL NITRIC OXIDE SYNTHASE GENE DISRUPTION REDUCES THE DIASTOLIC CALCIUM LEAK FROM THE RYANODINE RECEPTOR: IMPLICATIONS FOR EXCITATION-CONTRACTION COUPLING IN HEART FAILURE
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Nitric oxide synthases in heart failure.
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Protective vascular and cardiac effects of inducible nitric oxide synthase in mice with hyperhomocysteinemia.
ανά: Sanjana Dayal, κ.ά.
Έκδοση: (2014-01-01)

The subcellular localisation of neuronal nitric oxide synthase determines the downstream effects of NO on myocardial function
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Neuronal nitric oxide synthase gene transfer promotes cardiac vagal gain of function.
ανά: Mohan, R, κ.ά.
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Targeting neuronal nitric oxide synthase with gene transfer to modulate cardiac autonomic function.
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Targeting cardiac sympatho-vagal imbalance using gene transfer of nitric oxide synthase.
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Autocrine and paracrine nitric oxide regulate attachment of human osteoclasts.
ανά: Yaroslavskiy, B, κ.ά.
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MODELLING CALCIUM HANDLING MECHANISMS IN NORMAL AND NEURONAL NITRIC OXIDE SYNTHASE KNOCK-OUT MOUSE HEARTS
ανά: Li, L, κ.ά.
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The emerging role of neuronal nitric oxide synthase in the regulation of myocardial function.
ανά: Casadei, B
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Neuronal nitric oxide synthase gene transfer decreases [Ca2+]i in cardiac sympathetic neurons.
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Letter by Antoniades et al regarding article, "Uncoupled cardiac nitric oxide synthase mediates diastolic dysfunction".
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Neuronal nitric oxide synthase and human vascular regulation.
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Response of cardiac endothelial nitric oxide synthase to plasma viscosity modulation in acute isovolemic hemodilution
ανά: Kanyanatt Kanokwiroon, κ.ά.
Έκδοση: (2014-01-01)

Differential role of endothelial versus neuronal nitric oxide synthase in the regulation of coronary blood flow during pacing-induced increases in cardiac workload.
ανά: Shabeeh, H, κ.ά.
Έκδοση: (2013)

Inducible nitric oxide synthase-derived extracellular nitric oxide flux regulates proinflammatory responses at the single cell level
ανά: Veena Somasundaram, κ.ά.
Έκδοση: (2020-01-01)

Investigating cardiac energetics in heart failure.
ανά: Lygate, C, κ.ά.
Έκδοση: (2013)

Investigating cardiac energetics in heart failure
ανά: Lygate, C, κ.ά.
Έκδοση: (2013)

A cardiac-null mutation of Prdm16 causes hypotension in mice with cardiac hypertrophy via increased nitric oxide synthase 1.
ανά: Ji-One Kang, κ.ά.
Έκδοση: (2022-01-01)