Fast exocytosis by Ca2+-channel-granule interaction in mouse pancreatic B-cells

Fast exocytosis by Ca2+-channel-granule interaction in mouse pancreatic B-cells

Sonraí bibleagrafaíochta
Príomhchruthaitheoirí:	Renstrom, E, Atlas, D, Barg, S, Rorsman, P
Formáid:	Journal article
Foilsithe / Cruthaithe:	1999

Míreanna comhchosúla

Physical interaction between secretory granules and L-type calcium channels is required for fast exocytosis in B-cells
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Fast exocytosis with few Ca2+-channels by economical use of Ca2+ in insulin-secreting pancreatic B-cells
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Fast exocytosis with few Ca(2+) channels in insulin-secreting mouse pancreatic B cells.
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Fast insulin secretion reflects exocytosis of docked granules in mouse pancreatic B-cells.
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Intracellularly applied ion channel modulators interfere with calcium induced exocytosis in mouse pancreatic B-cells
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Cooling inhibits exocytosis in single mouse pancreatic B-cells by suppression of granule mobilization.
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Rapid ATP-dependent priming of secretory granules precedes Ca(2+)-induced exocytosis in mouse pancreatic B-cells.
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Co-localization of L-type Ca2+ channels and insulin-containing secretory granules and its significance for the initiation of exocytosis in mouse pancreatic B-cells.
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CaM kinase II-dependent mobilization of secretory granules underlies acetylcholine-induced stimulation of exocytosis in mouse pancreatic B-cells.
de réir: Gromada, J, et al.
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Regulation of insulin granule exocytosis
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Correlation between functional and ultrastructual measurements of exocytosis in mouse pancreatic B-cells
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ATP-dependence of exocytosis in mouse pancreatic beta-cells
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The stimulatory action of tolbutamide on Ca2+-dependent exocytosis is mediated by a 65-kDa mdr-like P-glycoprotein and ClC3Cl(-)-channels in granule membranes of insulin secreting B-cells.
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Priming of insulin granules for exocytosis by granular Cl(-) uptake and acidification.
de réir: Barg, S, et al.
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Intracellular tolbutamide stimulates calcium-dependent exocytosis in pancreatic B-cells
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Protein kinase A-dependent and -independent stimulation of exocytosis by cAMP in mouse pancreatic B-cells.
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A subset of 50 secretory granules in close contact with L-type Ca2+ channels accounts for first-phase insulin secretion in mouse beta-cells.
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Acetylcholine stimulates exocytosis by acceleration of granula mobilisation in mouse pancreatic B-cells
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RELATIONSHIP BETWEEN EXOCYTOSIS AND ENDOCYTOSIS IN PANCREATIC B-CELLS
de réir: Rorsman, P, et al.
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Adrenaline stimulates glucagon secretion in pancreatic A-cells by increasing the Ca2+ current and the number of granules close to the L-type Ca2+ channels.
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Sulfonylurea-mediated stimulation of insulin exocytosis via an ATP-sensitive K+ channel-independent action.
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Cyclic AMP stimulates insulin exocytosis by modification of granules already present at the release sites
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Activation of Ca(2+)-dependent K(+) channels contributes to rhythmic firing of action potentials in mouse pancreatic beta cells.
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Activation of Ca2+-dependent K+ channels contributes to rhythmic firing of action potentials in mouse pancreatic beta cells
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REGULATION OF EXOCYTOSIS IN THE PANCREATIC B-CELL - INTERACTIONS BETWEEN PKA AND PKC
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Exocytosis depends on granular chloride fluxes regulated by a sulfonylurea receptor-like protein in insulin-secreting B-cells
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Single Ca channel currents in mouse pancreatic B-cells.
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EXOCYTOSIS IN MOUSE PANCREATIC B-CELLS IS REGULATED BY THE BALANCE BETWEEN PHOSPHORYLATION AND DEPHOSPHORYLATION
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Large dense-core vesicle exocytosis in pancreatic beta-cells monitored by capacitance measurements.
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Probability of Exocytosis in Pancreatic β-Cells: Dependence on Ca2+ Sensing Latency Times, Ca2+ Channel Kinetic Parameters, and Channel Clustering
de réir: Galvanovskis, J, et al.
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Exocytosis from pancreatic β-cells: mathematical modelling of the exit of low-molecular-weight granule content.
de réir: Galvanovskis, J, et al.
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Insulin granule dynamics in pancreatic beta cells.
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SUR1 regulates PKA-independent cAMP-induced granule priming in mouse pancreatic B-cells.
de réir: Eliasson, L, et al.
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Ca(2+)- and GTP-dependent exocytosis in mouse pancreatic beta-cells involves both common and distinct steps.
de réir: Proks, P, et al.
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Cyclic AMP stimulates exocytosis in mouse B-cells by both PKA-dependent and PKA-independent mechanisms
de réir: Eliasson, L, et al.
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Tight coupling between electrical activity and exocytosis in mouse glucagon-secreting alpha-cells.
de réir: Barg, S, et al.
Foilsithe / Cruthaithe: (2000)

The stimulatory action of tolbutamide on Ca2+-dependent exocytosis in pancreatic beta cells is mediated by a 65-kDa mdr-like P-glycoprotein.
de réir: Barg, S, et al.
Foilsithe / Cruthaithe: (1999)

Role for [Ca2+] microdomains in glucose-induced movement of GFP-tagged secretory granules.
de réir: Renstrom, E, et al.
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Multivesicular exocytosis in rat pancreatic beta cells.
de réir: Hoppa, M, et al.
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CA-INDEPENDENT STIMULATION OF EXOCYTOSIS BY GTP IN SINGLE-MOUSE PANCREATIC BETA-CELLS
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