Risk factors for bronchial hyperresponsiveness in workers exposed to acid anhydrides.

Risk factors for bronchial hyperresponsiveness in workers exposed to acid anhydrides.

Risk factors for bronchial hyperresponsiveness have previously been established in the general community. In settings where occupational asthma is a risk, it has not been established whether occupational sensitization or measures of exposure are important. Bronchial responsiveness to histamine was m...

Bibliografske podrobnosti
Main Authors:	Barker, R, van Tongeren, M, Harris, J, Gardiner, K, Venables, K, Newman Taylor, A
Format:	Journal article
Jezik:	English
Izdano:	2000

Podobne knjige/članki

Risk factors for sensitisation and respiratory symptoms among workers exposed to acid anhydrides: a cohort study.
od: Barker, R, et al.
Izdano: (1998)

Risk factors for sensitisation and respiratory symptoms among workers exposed to acid anhydrides: A cohort study
od: Barker, R, et al.
Izdano: (1999)

Retrospective exposure assessment for a cohort study into respiratory effects of acid anhydrides.
od: van Tongeren, M, et al.
Izdano: (1998)

Exposure to acid anhydrides in three resin and one cushioned flooring manufacturing plants.
od: van Tongeren, M, et al.
Izdano: (1995)

Apoptosis in resolution of bronchial hyperresponsiveness
od: Heinzmann Andrea, et al.
Izdano: (2001-03-01)

Acid anhydrides and asthma.
od: Taylor, A, et al.
Izdano: (1987)

Occupational asthma caused by tetrachlorophthalic anhydride: a 12-year follow-up.
od: Barker, R, et al.
Izdano: (1998)

Exposure-response relationships in asthma caused by tetrachlorophthalic anhydride.
od: Venables, K, et al.
Izdano: (1990)

Airway hyperresponsiveness in bronchial asthma: pathogenesis basics
od: I. A. Deyev, et al.
Izdano: (2002-12-01)

Bronchial Hyperresponsiveness in Patients with Inflammatory Bowel Disease
od: G. Ilonidis, et al.
Izdano: (2004-09-01)

Occupational asthma caused by tetrachlorophthalic anhydride: A 12-year follow-up
od: Barker, R, et al.
Izdano: (1998)

Low molecular weight chemicals, hypersensitivity, and direct toxicity: the acid anhydrides.
od: Venables, K
Izdano: (1989)

Specificity of the human IgE response to inhaled acid anhydrides.
od: Topping, MD, et al.
Izdano: (1986)

Exposure-response relationships in asthma caused by tetrachlorophthalic anhydride
od: Venables, K, et al.
Izdano: (1990)

Does Airway T2 Inflammation Increase the Severity of Bronchial Hyperresponsiveness?
od: Miguel Jiménez-Gómez, et al.
Izdano: (2023-10-01)

Cross-sectional study of cholinergic urticaria subtypes and bronchial hyperresponsiveness
od: Naoko Katsurada, et al.
Izdano: (2022-10-01)

Bronchial hyperresponsiveness and analysis of induced sputum cells in Crohn's disease
od: R.M. Bartholo, et al.
Izdano: (2005-02-01)

A Surrogate Marker of Airway Hyperresponsiveness in Patients with Bronchial Asthma
od: Yuta Kono, et al.
Izdano: (2014-01-01)

Sex-based differences in factors associated with bronchial hyperresponsiveness in adolescents with childhood asthma
od: Young Hwan Kim, et al.
Izdano: (2021-05-01)

Th9 cells induce steroid-resistant bronchial hyperresponsiveness in mice
od: Mayumi Saeki, et al.
Izdano: (2017-09-01)

Association of HLA-DQ5 and HLA-DR1 with sensitization to organic acid anhydrides.
od: Jones, MG, et al.
Izdano: (2004)

Increase of Exhaled Nitric Oxide (eNO) after Methylene Diphenyl Diisocyanate (MDI) Exposure in Isocyanate Workers with Bronchial Hyperresponsiveness
od: Xaver Baur, et al.
Izdano: (2005-01-01)

Role of bronchial hyperresponsiveness in patients with obstructive sleep apnea with asthma-like symptoms
od: Akiko Sano, et al.
Izdano: (2024-04-01)

Bronchial hyperresponsiveness and asthma during oral immunotherapy for egg or peanut allergy in children
od: Janne Burman, et al.
Izdano: (2022-10-01)

Bronchial hyperresponsiveness, airway inflammation, and reversibility in patients with chronic obstructive pulmonary disease
od: Zanini A, et al.
Izdano: (2015-06-01)

Bronchial hyperresponsiveness in women with chronic obstructive pulmonary disease related to wood smoke
od: Jaramillo C, et al.
Izdano: (2012-06-01)

Immunologic and functional consequences of chemical (tetrachlorophthalic anhydride)-induced asthma after four years of avoidance of exposure.
od: Venables, K, et al.
Izdano: (1987)

Exposure-response relations among laboratory animal workers exposed to rats.
od: Nieuwenhuijsen, M, et al.
Izdano: (2003)

Bronchial granulocytes in the development of epithelial destruction and oxidative lipid modification in patients with bronchial asthma with cold and osmotic airway hyperresponsiveness
od: A. B. Pirogov, et al.
Izdano: (2021-04-01)

Bronchial provocation tests: The rationale for using inhaled mannitol as a test for airway hyperresponsiveness
od: D Brannan, et al.
Izdano: (2002-04-01)

Role of neutral endopeptidase in endotoxin-induced bronchial hyperresponsiveness to substance P in guinea pigs
od: Shigenori Iwamae, et al.
Izdano: (1998-01-01)

Stepwise health surveillance for bronchial irritability syndrome in workers at risk of occupational respiratory disease.
od: Post, W, et al.
Izdano: (1998)

The Particularities of Airway Hyperresponsiveness to Histamine in Bronchial Asthma School-Age Children in Different Inflammatory Phenotypes
od: Ye.P. Ortemenka
Izdano: (2015-10-01)

Tetrachlorophthalic anhydride asthma: Evidence for specific IgE antibody
od: Venables, K, et al.
Izdano: (1982)

Efficacy of measures of hygiene in workers sensitised to acid anhydrides and the influence of selection bias on the results.
od: Drexler, H, et al.
Izdano: (1999)

Green trimellitic anhydride and hemimellitic anhydride from malic acid
od: Aleksei Ananin, et al.
Izdano: (2023-01-01)

Tetrachlorophthalic anhydride asthma: evidence for specific IgE antibody.
od: Howe, W, et al.
Izdano: (1983)

Tetrachlorophthalic anhydride asthma: evidence for specific IgE antibody
od: Howe, W, et al.
Izdano: (1983)

Work related symptoms, sensitisation, and estimated exposure in workers not previously exposed to flour.
od: Cullinan, P, et al.
Izdano: (1994)

Destruction of the bronchial epithelium in patients with severe asthma according to different patterns of inflammation and cold airway hyperresponsiveness
od: A B Pirogov, et al.
Izdano: (2019-03-01)