Protective effect of prior fimbria-fornix deafferentation on hippocampal CA1 following transient forebrain ischemia is mediated by GluR2 expression

Protective effect of prior fimbria-fornix deafferentation on hippocampal CA1 following transient forebrain ischemia is mediated by GluR2 expression

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Kaur, J, Zhao, Z, Geransar, R, Sun, P, Buchan, A
التنسيق:	Conference item
منشور في:	2005

مواد مشابهة

Prior deafferentation confers long term protection to CA1 against transient forebrain ischemia and sustains GluR2 expression.
حسب: Kaur, J, وآخرون
منشور في: (2006)

Neuroprotection and neurogenesis: modulation of cornus ammonis 1 neuronal survival after transient forebrain ischemia by prior fimbria-fornix deafferentation.
حسب: Zhao, Z, وآخرون
منشور في: (2006)

Fimbria/Fornix deafferentation protects CA1 neurons and enhances neurogenesis in hippocampus following global ischemia
حسب: Zhao, Z, وآخرون
منشور في: (2003)

Hypothermia rescues hippocampal CA1 neurons and attenuates down-regulation of the AMPA receptor GluR2 subunit after forebrain ischemia.
حسب: Colbourne, F, وآخرون
منشور في: (2003)

The effects of hippocampal and fimbria-fornix lesions on prepulse inhibition.
حسب: Pouzet, B, وآخرون
منشور في: (1999)

Blockade of the AMPA receptor prevents CA1 hippocampal injury following severe but transient forebrain ischemia in adult rats.
حسب: Buchan, A, وآخرون
منشور في: (1991)

Septo-hippocampal deafferentation protects CA1 neurons against ischemic injury.
حسب: Buchan, A, وآخرون
منشور في: (1990)

Systematic comparison of the effects of hippocampal and fornix-fimbria lesions on acquisition of three configural discriminations
حسب: McDonald, R, وآخرون
منشور في: (1997)

The role of the GluR-A (GluR1) AMPA receptor subunit in learning and memory.
حسب: Sanderson, D, وآخرون
منشور في: (2008)

Fornix-fimbria section and the partial reinforcement extinction effect.
حسب: Feldon, J, وآخرون
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Conformational dynamics of glutamate receptors: MD simulations of the ligand-binding domains of GluR2 and GluR0
حسب: Arinaminpathy, Y, وآخرون
منشور في: (2004)

The effects of hippocampal formation ablation or fimbria-fornix section on performance of a nonspatial radial arm maze task by rats.
حسب: Yee, B, وآخرون
منشور في: (1994)

The time course of the hyperactivity that follows lesions or temporary inactivation of the fimbria-fornix.
حسب: Bannerman, D, وآخرون
منشور في: (2001)

Septal transplants restore maze learning in rats with fornix-fimbria lesions.
حسب: Dunnett, S, وآخرون
منشور في: (1982)

Induction and expression of GluA1 (GluR-A)-independent LTP in the hippocampus.
حسب: Romberg, C, وآخرون
منشور في: (2009)

HIPPOCAMPAL DEAFFERENTATION - TRANSPLANT DERIVED REINNERVATION AND FUNCTIONAL RECOVERY
حسب: Dunnett, S, وآخرون
منشور في: (1982)

HIPPOCAMPAL DEAFFERENTATION - TRANSPLANT-DERIVED REINNERVATION AND FUNCTIONAL RECOVERY
حسب: Dunnett, S, وآخرون
منشور في: (1984)

Interaction of perirhinal cortex with the fornix-fimbria: memory for objects and "object-in-place" memory.
حسب: Gaffan, D, وآخرون
منشور في: (1996)

Spatial memory dissociations in mice lacking GluR1.
حسب: Reisel, D, وآخرون
منشور في: (2002)

In vivo binding of [3H]nimodipine in rat brain after transient forebrain ischemia.
حسب: Takizawa, S, وآخرون
منشور في: (1994)

Fornix-fimbria section and working memory deficits in rats: stimulus complexity and stimulus size.
حسب: Cassaday, H, وآخرون
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Edited GluR2, a gatekeeper for motor neurone survival?
حسب: Buckingham, S, وآخرون
منشور في: (2008)

Spatial reference memory in GluR-A-deficient mice using a novel hippocampal-dependent paddling pool escape task.
حسب: Schmitt, W, وآخرون
منشور في: (2004)

Erythropoietin Improves Place Learning in an 8-Arm Radial Maze in Fimbria-Fornix Transected Rats
حسب: Hana Malá, وآخرون
منشور في: (2005-01-01)

Extracellular interactions between GluR2 and N-cadherin in spine regulation.
حسب: Saglietti, L, وآخرون
منشور في: (2007)

Novel apoptotic evidence for delayed neuronal death in the hippocampal CA1 pyramidal cells after transient ischemia - Response
حسب: Colbourne, F, وآخرون
منشور في: (2000)

The drugs don't work-or do they? Pharmacological and transgenic studies of the contribution of NMDA and GluR-A-containing AMPA receptors to hippocampal-dependent memory.
حسب: Bannerman, D, وآخرون
منشور في: (2006)

Treatment with an AMPA antagonist 12 hours following severe normothermic forebrain ischemia prevents CA1 neuronal injury.
حسب: Li, H, وآخرون
منشور في: (1993)

GluR-A-dependent synaptic plasticity is required for the temporal encoding of nonspatial information.
حسب: Reisel, D, وآخرون
منشور في: (2005)

Absence of antibodies to glutamate receptor type 3 (GluR3) in Rasmussen encephalitis.
حسب: Watson, R, وآخرون
منشور في: (2004)

Restoration of spatial working memory by genetic rescue of GluR-A-deficient mice.
حسب: Schmitt, W, وآخرون
منشور في: (2005)

Indefatigable CA1 sector neuroprotection with mild hypothermia induced 6 hours after severe forebrain ischemia in rats.
حسب: Colbourne, F, وآخرون
منشور في: (1999)

Molecular dynamics simulations of the ligand-binding domain of the ionotropic glutamate receptor GluR2.
حسب: Arinaminpathy, Y, وآخرون
منشور في: (2002)

A prokaryotic glutamate receptor: homology modelling and molecular dynamics simulations of GluR0.
حسب: Arinaminpathy, Y, وآخرون
منشور في: (2003)

Homology modelling and molecular dynamics simulations of a prokaryotic glutamate receptor: GluR0
حسب: Arinaminpathy, Y, وآخرون
منشور في: (2002)

A selective N-type Ca(2+)-channel blocker prevents CA1 injury 24 h following severe forebrain ischemia and reduces infarction following focal ischemia.
حسب: Buchan, A, وآخرون
منشور في: (1994)

Upregulation of AMPA receptor GluR2 (GluA2) subunits in subcortical ischemic vascular dementia is repressed in the presence of Alzheimer's disease.
حسب: Mohamed, N, وآخرون
منشور في: (2011)

Upregulation of AMPA receptor GluR2 (GluA2) subunits in subcortical ischemic vascular dementia is repressed in the presence of Alzheimer's disease
حسب: Mohamed, N, وآخرون
منشور في: (2011)

The correlation of GluR3B antibody with T lymphocyte subsets and inflammatory factors and their role in the progression of epilepsy
حسب: Qingwei Lai, وآخرون
منشور في: (2024-09-01)

The N-methyl-D-aspartate antagonist, MK-801, fails to protect against neuronal damage caused by transient, severe forebrain ischemia in adult rats.
حسب: Buchan, A, وآخرون
منشور في: (1991)