Oxidative stress in endometriosis induces IL-8 release directly via 4-HNE
Lipid peroxidation products, e.g. 4-hydroxynonenal (4-HNE), are generated by oxidative stress, and are suggested to cause endometriosis-associated pain following iron overload after retrograde menstrual bleeding. Our aim was to determine the role of oxidative stress including 4-HNE protein adduct fo...
| Main Authors: | , , , , , , , , , |
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| Format: | Conference item |
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SAGE Publications
2018
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| _version_ | 1797100338677809152 |
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| author | Elger, B Mueller, J Morton, K Kessler, B Becker, C Steers, G Thezenas, M Zondervan, K Zollner, T Oppermann, U |
| author_facet | Elger, B Mueller, J Morton, K Kessler, B Becker, C Steers, G Thezenas, M Zondervan, K Zollner, T Oppermann, U |
| author_sort | Elger, B |
| collection | OXFORD |
| description | Lipid peroxidation products, e.g. 4-hydroxynonenal (4-HNE), are generated by oxidative stress, and are suggested to cause endometriosis-associated pain following iron overload after retrograde menstrual bleeding. Our aim was to determine the role of oxidative stress including 4-HNE protein adduct formation in endometriotic lesions and in peritoneal fluid from endometriosis patients; and characterise the proinflammatory effects caused by 4-HNE in endometriosis. |
| first_indexed | 2024-03-07T05:36:04Z |
| format | Conference item |
| id | oxford-uuid:e3ee8f83-1d58-4741-bd6e-c91d9a5d55cb |
| institution | University of Oxford |
| last_indexed | 2024-03-07T05:36:04Z |
| publishDate | 2018 |
| publisher | SAGE Publications |
| record_format | dspace |
| spelling | oxford-uuid:e3ee8f83-1d58-4741-bd6e-c91d9a5d55cb2022-03-27T10:12:49ZOxidative stress in endometriosis induces IL-8 release directly via 4-HNEConference itemhttp://purl.org/coar/resource_type/c_5794uuid:e3ee8f83-1d58-4741-bd6e-c91d9a5d55cbSymplectic Elements at OxfordSAGE Publications2018Elger, BMueller, JMorton, KKessler, BBecker, CSteers, GThezenas, MZondervan, KZollner, TOppermann, ULipid peroxidation products, e.g. 4-hydroxynonenal (4-HNE), are generated by oxidative stress, and are suggested to cause endometriosis-associated pain following iron overload after retrograde menstrual bleeding. Our aim was to determine the role of oxidative stress including 4-HNE protein adduct formation in endometriotic lesions and in peritoneal fluid from endometriosis patients; and characterise the proinflammatory effects caused by 4-HNE in endometriosis. |
| spellingShingle | Elger, B Mueller, J Morton, K Kessler, B Becker, C Steers, G Thezenas, M Zondervan, K Zollner, T Oppermann, U Oxidative stress in endometriosis induces IL-8 release directly via 4-HNE |
| title | Oxidative stress in endometriosis induces IL-8 release directly via 4-HNE |
| title_full | Oxidative stress in endometriosis induces IL-8 release directly via 4-HNE |
| title_fullStr | Oxidative stress in endometriosis induces IL-8 release directly via 4-HNE |
| title_full_unstemmed | Oxidative stress in endometriosis induces IL-8 release directly via 4-HNE |
| title_short | Oxidative stress in endometriosis induces IL-8 release directly via 4-HNE |
| title_sort | oxidative stress in endometriosis induces il 8 release directly via 4 hne |
| work_keys_str_mv | AT elgerb oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT muellerj oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT mortonk oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT kesslerb oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT beckerc oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT steersg oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT thezenasm oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT zondervank oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT zollnert oxidativestressinendometriosisinducesil8releasedirectlyvia4hne AT oppermannu oxidativestressinendometriosisinducesil8releasedirectlyvia4hne |