Plasma levels of sCD40-ligand interact with homocysteine biosynthetic pathway and predict eNOS coupling in human vessels: effects on vascular redox state

Bibliographic Details
Main Authors:	Antoniades, C, Shirodaria, C, Baarholm, O, Leeson, P, Antonopoulos, A, Pillai, R, Tousoulis, D, Refsum, H, Stefanadis, C, Channon, K
Format:	Journal article
Published:	2008

Soluble CD40-ligand levels are associated with vascular superoxide production and eNOS coupling in human vessels: Interrelationships with homocysteine biosynthetic pathway
by: Antoniades, C, et al.
Published: (2008)

Vascular Homocysteine Versus 5-Methyl-Tetrahydrofolate As Regulators of Vascular Redox and Endothelial Function in Human Vessels
by: Antoniades, C, et al.
Published: (2009)

Vascular Homocysteine vs 5-Methyl-Tetrahydrofolate as Regulators of Vascular Redox and Endothelial Function in Human Vessels
by: Antoniades, C, et al.
Published: (2008)

Genetic variability of MTHFR affects vascular redox and nitric oxide bioavailability in human vessels, by modifying eNOS coupling
by: Antoniades, C, et al.
Published: (2008)

MTHFR 677 C>T Polymorphism reveals functional importance for 5-methyltetrahydrofolate, not homocysteine, in regulation of vascular redox state and endothelial function in human atherosclerosis.
by: Antoniades, C, et al.
Published: (2009)

Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling
by: Antoniades, C, et al.
Published: (2008)

Plasma asymmetrical-dimethyl-arginine (ADMA) as a regulator of vascular redox and nitric oxide bioavailability in human vessels
by: Antoniades, C, et al.
Published: (2007)

A Novel Haplotype on GCH1 Gene, Encoding GTP Cyclohydrolase 1, Regulates Vascular Biopterin Synthesis, eNOS Coupling and Vascular Redox in Human Vessels
by: Antoniades, C, et al.
Published: (2008)

Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
by: Antoniades, C, et al.
Published: (2009)

Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
by: Antoniades, C, et al.
Published: (2008)

The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
by: Antoniades, C, et al.
Published: (2007)

Pre-operative plasma sCD40L, endothelial function and vascular redox state predict the development of atrial fibrillation
by: Antoniades, C, et al.
Published: (2008)

Homocysteine lowering: any use in atherosclerosis?
by: Antoniades, C, et al.
Published: (2007)

Preoperative Circulating sCD40L And Vascular Redox State in LIMA Grafts, Predict The Development Of Atrial Fibrillation Post-Coronary Artery Bypass Grafting
by: Antoniades, C, et al.
Published: (2009)

Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels
by: Antoniades, C, et al.
Published: (2007)

Genetic Variability of Methyl-Tetrahydrofolate-Reductase Modifies eNOS Coupling in Human Arteries and Veins: Effects on Vascular Superoxide Generation and Nitric Oxide Bioavailabllity
by: Antoniades, C, et al.
Published: (2008)

Pre-Operative Estimation of Endothelial Function and Plasma sCD40L as Well as Vascular Redox State, Predict the Development of Atrial Fibrillation Post-Coronary Artery Bypass Grafting Operation
by: Antoniades, C, et al.
Published: (2008)

5-methyl-tetrahydrofolate increases eNOS activity by preventing peroxynitrite-induced oxidation of tetrahydrobiopterin in human vessels
by: Antoniades, C, et al.
Published: (2006)

5-Methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production by improving eNOS coupling and increasing tetrahydrobiopterin bioavailability in human vessels in vivo
by: Antoniades, C, et al.
Published: (2006)

5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
by: Antoniades, C, et al.
Published: (2006)

Atorvastatin rapidly improves redox state in human vein grafts ex-vivo, by directly reducing NADPH-oxidase activity and improving eNOS coupling
by: Bakogiannis, C, et al.
Published: (2010)

Preoperative sCD40L levels predict risk of atrial fibrillation after off-pump coronary artery bypass graft surgery.
by: Antoniades, C, et al.
Published: (2009)

Intravenous infusion of 5-methyl-tetrahydrofolate rapidly improves eNOS Coupling. In human vessels by increasing tetrahydrobiopterin bioavailability
by: Antoniades, C, et al.
Published: (2006)

Oral Atorvastatin Treatment Improves NO Bioavailability and Vascular Redox State in the Human Arterial Wall, via a Tetrahydrobiopterin-Mediated Improvement of eNOS Coupling
by: Antoniades, C, et al.
Published: (2011)

5-methyl-tetrahydrofolate increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling and preventing the intracellular oxidation of tetrahy-drobiopterin, in human vessels in vivo
by: Antoniades, C, et al.
Published: (2006)

Homocysteine and coronary atherosclerosis: from folate fortification to the recent clinical trials.
by: Antoniades, C, et al.
Published: (2009)

Redox state in arterial grafts predicts left ventricular functional restoration after coronary artery bypass grafting
by: Antoniades, C, et al.
Published: (2009)

Atorvastatin directly improves vascular redox state by affecting eNOS coupling and NADPH-oxidase activity in human vein grafts ex-vivo
by: Bakogiannis, C, et al.
Published: (2010)

Low- and high- dose treatment with folic acid, equally improve endothelial function and decrease superoxide production in human vessels via tetrahydrobiopterin-mediated eNOS coupling
by: Antoniades, C, et al.
Published: (2007)

Chronic treatment with folic acid improves nitric oxide bioavailability and decreases superoxide production in human vessels, through tetrahydrobiopterin-mediated improvement of eNOS coupling
by: Antoniades, C, et al.
Published: (2007)

Atorvastatin rapidly modifies vascular redox state and improves nitric oxide bioavailability in human vessels obtained from patients with advanced atherosclerosis
by: Demosthenous, M, et al.
Published: (2010)

RAPID, DIRECT EFFECTS OF STATIN TREATMENT ON ARTERIAL REDOX STATE AND NITRIC OXIDE BIOAVAILABILITY IN HUMAN ATHEROSCLEROSIS VIA TETRAHYDROBIOPTERIN-MEDIATED ENOS COUPLING
by: Antoniades, C, et al.
Published: (2011)

Rapid Effects of Atorvastatin on Tetrahydrobiopterin-Mediated eNOS Coupling in Human Arteries, is Mediated by Stimulation of Vascular GTP-Cyclohydrolase
by: Antoniades, C, et al.
Published: (2011)

The CD40/CD40 ligand system: linking inflammation with atherothrombosis.
by: Antoniades, C, et al.
Published: (2009)

Associations Between Serum Adiponectin, Vascular O2-Generation and NO Bioavailability In Patients With Coronary Atherosclerosis
by: Antoniades, C, et al.
Published: (2010)

Serum adiponectin regulates vascular O2-generation and NO bioavailability in patients with coronary atherosclerosis
by: Antoniades, C, et al.
Published: (2010)

Association of plasma asymmetrical dimethylarginine (ADMA) with elevated vascular superoxide production and endothelial nitric oxide synthase uncoupling: implications for endothelial function in human atherosclerosis.
by: Antoniades, C, et al.
Published: (2009)

Short-Term Atorvastatin Treatment Reduces Vascular Oxidative Stress Saphenous Vein Grafts by Improving eNOS Coupling and Reducing NADPH-Oxidase Activity in Patients Undergoing Elective CABG
by: Antoniades, C, et al.
Published: (2009)

Novel therapeutic strategies targeting vascular redox in human atherosclerosis.
by: Antonopoulos, A, et al.
Published: (2009)

Evaluation of endothelial function
by: Antoniades, C, et al.
Published: (2011)

Plasma levels of sCD40-ligand interact with homocysteine biosynthetic pathway and predict eNOS coupling in human vessels: effects on vascular redox state

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