Asymmetrical dimethylarginine as a mediator of endothelial dysfunction in acute inflammation at different disease stages

ग्रंथसूची विवरण
मुख्य लेखकों:	Demosthenous, M, Antoniades, C, Antonopoulos, S, Tousoulis, D, Bakogiannis, C, Vlachopoulos, C, Petrochilou, K, Toutouza, M, Channon, K, Stefanadis, C
स्वरूप:	Conference item
प्रकाशित:	2010

Role of asymmetrical dimethylarginine in inflammation-induced endothelial dysfunction in human atherosclerosis.
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

Haplotype on GTP-Cyclohydrolase I Gene, Reveals a Novel Role of Tetrahydrobiopterin as a Defense Mechanism Against Inflammation-Induced Endothelial Dysfunction in Human Atherosclerosis
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

Response to Asymmetrical Dimethylarginine, Oxidative Stress, and Atherosclerosis
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

NADPH-oxidase activity in peripheral blood mononuclear cells as a novel inflammatory marker in atherosclerosis
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2010)

Induction of vascular GTP-cyclohydrolase I and endogenous tetrahydrobiopterin synthesis protect against inflammation-induced endothelial dysfunction in human atherosclerosis.
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

Induction of vascular GTP-cyclohydrolase i and endogenous tetrahydrobiopterin synthesis protect against inflammation-induced endothelial dysfunction in human atherosclerosis
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

Association of plasma asymmetrical dimethylarginine (ADMA) with elevated vascular superoxide production and endothelial nitric oxide synthase uncoupling: implications for endothelial function in human atherosclerosis.
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2009)

Acute Inflammation Rapidly Increases Plasma Biopterins and Impairs Endothelial Function: A Novel Role of Plasma Tetrahydrobiopterin as a Marker of Acute Inflammation
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2011)

Methionine-induced homocysteinemia impairs endothelial function in hypertensives: the role of asymmetrical dimethylarginine and antioxidant vitamins.
द्वारा: Tousoulis, D, और अन्य
प्रकाशित: (2011)

Asymmetrical dimethylarginine regulates endothelial function in methionine-induced but not in chronic homocystinemia in humans: effect of oxidative stress and proinflammatory cytokines.
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2006)

Platelet activation in atherogenesis associated with low-grade inflammation.
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2010)

Statin treatment improves vascular redox state through a tetrahydrobiopterin-mediated re-coupling of endothelial nitric oxide synthase, in human atherosclerosis
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

The role of adiponectin at different stages of atherosclerosis in heart failure: associations with myocardial redox state
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2010)

Adiponectin as a Biomarker at Different Stages of Ischaemic Heart Disease: From Obesity to Ischaemic Heart Failure
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2009)

Evaluation of endothelial function
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

The CD40/CD40 ligand system: linking inflammation with atherothrombosis.
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2009)

Gene therapy for dysfunctional endothelium: is dimethylarginine dimethylaminohydrolase-2 a therapeutic target?
द्वारा: Tousoulis, D, और अन्य
प्रकाशित: (2010)

Study of endothelial dysfunction and asymmetric dimethylarginine levels
द्वारा: Ya. V. Sirman, और अन्य
प्रकाशित: (2019-10-01)

Genetic variability on adiponectin gene affects myocardial infarction risk: the role of endothelial dysfunction.
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2013)

Genetic variability on adiponectin gene affects myocardial infarction risk: The role of endothelial dysfunction
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2013)

Asymmetric dimethylarginine (ADMA) and endothelial dysfunction: implications for atherogenesis
द्वारा: Maurício Batista Paes Landim, और अन्य
प्रकाशित: (2009-05-01)

Novel therapeutic strategies targeting vascular redox in human atherosclerosis.
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2009)

Mechanisms of testosterone deficiency-related endothelial dysfunction: invited commentary for the Hellenic Journal of Cardiology on: Tsikas et al. “Associations between asymmetric dimethylarginine, nitrite-dependent renal carbonic anhydrase activity and plasma testosterone levels in hypogonadal men”
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2018)

Short-Term Atorvastatin Treatment Reduces Vascular Oxidative Stress Saphenous Vein Grafts by Improving eNOS Coupling and Reducing NADPH-Oxidase Activity in Patients Undergoing Elective CABG
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2009)

Atorvastatin rapidly improves redox state in human vein grafts ex-vivo, by directly reducing NADPH-oxidase activity and improving eNOS coupling
द्वारा: Bakogiannis, C, और अन्य
प्रकाशित: (2010)

Atorvastatin rapidly modifies vascular redox state and improves nitric oxide bioavailability in human vessels obtained from patients with advanced atherosclerosis
द्वारा: Demosthenous, M, और अन्य
प्रकाशित: (2010)

Myocardial Redox State Predicts Post-Operative Clinical Outcome in Patients Undergoing Elective CABG: The Role of NADPH-Oxidase and Uncoupled Nitric Oxide Synthase
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2010)

Interrelationships between vascular superoxide and adiponectin biosynthesis in perivascular adipose tissue in human atherosclerosis
द्वारा: Bakogiannis, C, और अन्य
प्रकाशित: (2010)

Genetic Variability of Adiponectin Gene Defines the Biosynthesis Rate of Adiponectin in Adipose Tissue and Regulates Superoxide Generation in Human Arteries in vivo
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2010)

Associations Between Serum Adiponectin, Vascular O2-Generation and NO Bioavailability In Patients With Coronary Atherosclerosis
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2010)

Serum adiponectin regulates vascular O2-generation and NO bioavailability in patients with coronary atherosclerosis
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2010)

Rapid Effects of Atorvastatin on Tetrahydrobiopterin-Mediated eNOS Coupling in Human Arteries, is Mediated by Stimulation of Vascular GTP-Cyclohydrolase
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

Oral Atorvastatin Treatment Improves NO Bioavailability and Vascular Redox State in the Human Arterial Wall, via a Tetrahydrobiopterin-Mediated Improvement of eNOS Coupling
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

Preoperative atorvastatin treatment rapidly improves vein graft redox state by inhibiting rac1 and NADPH oxidase activity in patients undergoing elective CABG
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2010)

Atorvastatin directly improves vascular redox state by affecting eNOS coupling and NADPH-oxidase activity in human vein grafts ex-vivo
द्वारा: Bakogiannis, C, और अन्य
प्रकाशित: (2010)

Hyperuricemia induces endothelial dysfunction and accelerates atherosclerosis by disturbing the asymmetric dimethylarginine/dimethylarginine dimethylaminotransferase 2 pathway
द्वारा: Tzong-Shyuan Lee, और अन्य
प्रकाशित: (2021-10-01)

LOCAL ADIPONECTIN SYNTHESIS IN ADIPOSE TISSUE REGULATES VASCULAR SUPEROXIDE GENERATION IN PATIENTS WITH CORONARY ATHEROSCLEROSIS: INSIGHTS BY USING FUNCTIONAL GENETIC POLYMORPHISMS OF ADIPONECTIN GENE
द्वारा: Antoniades, C, और अन्य
प्रकाशित: (2011)

Effects of atorvastatin on endothelial function and the expression of proinflammatory cytokines and adhesion molecules in young subjects with successfully repaired coarctation of aorta.
द्वारा: Brili, S, और अन्य
प्रकाशित: (2012)

Effects of atorvastatin on endothelial function and the expression of proinflammatory cytokines and adhesion molecules in young subjects with successfully repaired coarctation of aorta
द्वारा: Brili, S, और अन्य
प्रकाशित: (2012)

Differential responses of distinct adipose tissue depots to acute and chronic inflammation: novel insights into the complex mechanisms regulating adiponectin biosynthesis in humans
द्वारा: Antonopoulos, A, और अन्य
प्रकाशित: (2013)

Asymmetrical dimethylarginine as a mediator of endothelial dysfunction in acute inflammation at different disease stages

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