Interleukin-23 drives intestinal inflammation through direct activity on T cells

Interleukin-23 drives intestinal inflammation through direct activity on T cells

Εμφάνιση άλλων εκδόσεων (2)

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Schiering, C, Ahern, P, Maloy, K, Powrie, F
Μορφή:	Conference item
Έκδοση:	2010

Παρόμοια τεκμήρια

Interleukin-23 drives intestinal inflammation through direct activity on T cells.
ανά: Ahern, P, κ.ά.
Έκδοση: (2010)

Interleukin-23 drives intestinal inflammation through direct activity on T cells
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Έκδοση: (2011)

Interleukin-23 Drives Intestinal Inflammation through Direct Activity on T Cells (vol 33, pg 279, 2010)
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The interleukin-23 axis in intestinal inflammation.
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Interleukin-23 drives innate and T cell-mediated intestinal inflammation.
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The Interleukin-23 / Interleukin-17 axis in intestinal inflammation.
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Innate lymphoid cells drive interleukin-23-dependent innate intestinal pathology.
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Dysregulated hematopoietic stem and progenitor cell activity promotes interleukin-23-driven chronic intestinal inflammation.
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Dysregulated Hematopoietic Stem and Progenitor Cell Activity Promotes Interleukin-23-Driven Chronic Intestinal Inflammation
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Interleukin-23 restrains regulatory T cell activity to drive T cell-dependent colitis.
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CD4(+) T-cell subsets in intestinal inflammation.
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Control of intestinal inflammation by regulatory T cells
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Identification of the cellular and molecular mechanisms of IL-23 driven intestinal inflammation
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MyD88 signalling from distinct cellular compartments drives intestinal inflammation
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An essential role for interleukin 10 in the function of regulatory T cells that inhibit intestinal inflammation
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An essential role for interleukin 10 in the function of regulatory T cells that inhibit intestinal inflammation.
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IL-1 mediates intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4+Th17 cells
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Control of intestinal inflammation by regulatory T cells.
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Characterisation of innate lymphoid cells that drive IL-23-dependent intestinal pathology
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TLR2-independent induction and regulation of chronic intestinal inflammation.
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IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4(+) Th17 cells.
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Characterization of the role of IL-10 in resolution from intestinal inflammation
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IL-23 production by CD11c(+) mononuclear phagocytes drives chronic Helicobacter hepaticus-induced intestinal inflammation
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Exacerbated hematopoietic stem and progenitor cell activity during intestinal inflammation
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Έκδοση: (2011)

T-bet is a key modulator of IL-23-driven pathogenic CD4(+) T cell responses in the intestine
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T-bet is a key modulator of IL-23-driven pathogenic CD4+ T cell responses in the intestine
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Έκδοση: (2016)

Regulatory lymphocytes and intestinal inflammation.
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Έκδοση: (2009)

Regulatory T cells suppress systemic and mucosal immune activation to control intestinal inflammation.
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Control of intestinal inflammation by regulatory T cells.
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IL-23 and Th17 cytokines in intestinal homeostasis.
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Έκδοση: (2008)

Regulatory T cells and intestinal homeostasis.
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The role of mucosal T lymphocytes in regulating intestinal inflammation.
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A GM-CSF eosinophil axis promotes IL-23-mediated chronic intestinal inflammation
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Intestinal inflammation abrogates the tolerogenic properties of MLN CD103+ dendritic cells.
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NLRC4 mediates protection in bacterially triggered intestinal inflammation
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Intestinal homeostasis and its breakdown in inflammatory bowel disease.
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NLR expression during the induction and resolution of intestinal inflammation
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Correction: Interleukin-4 and interleukin-10 synergize to inhibit cell-mediated immunity in vivo (European Journal of Immunology (1993) 23 9 (2226))
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Animal models of intestinal inflammation: clues to the pathogenesis of inflammatory bowel disease.
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