B-POCKET TRANSPLANT FROM HLA-A2 INTO B27 CHANGES REQUIREMENT FOR P-2 ANCHOR RESIDUE IN PEPTIDE PRESENTATION

B-POCKET TRANSPLANT FROM HLA-A2 INTO B27 CHANGES REQUIREMENT FOR P-2 ANCHOR RESIDUE IN PEPTIDE PRESENTATION

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Colbert, R, Rowlandjones, S, Mcmichael, A, Frelinger, J
التنسيق:	Journal article
منشور في:	1993

مواد مشابهة

Allele-specific B pocket transplant in class I major histocompatibility complex protein changes requirement for anchor residue at P2 of peptide.
حسب: Colbert, R, وآخرون
منشور في: (1993)

Allele-specific B pocket transplant in class I major histocompatibility complex protein changes requirement for anchor residue at P2 of peptide.
حسب: Colbert, R, وآخرون
منشور في: (1993)

DIFFERENCES IN PEPTIDE PRESENTATION BETWEEN B27 SUBTYPES - THE IMPORTANCE OF THE P1 SIDE-CHAIN IN MAINTAINING HIGH-AFFINITY PEPTIDE BINDING TO B-ASTERISK-2703
حسب: Colbert, R, وآخرون
منشور في: (1994)

Structural homologies between two HLA B27-restricted peptides suggest residues important for interaction with HLA B27.
حسب: Huet, S, وآخرون
منشور في: (1990)

Differences in peptide presentation between B27 subtypes: the importance of the P1 side chain in maintaining high affinity peptide binding to B*2703.
حسب: Colbert, R, وآخرون
منشور في: (1994)

Naturally occurring A pocket polymorphism in HLA-B*2703 increases the dependence on an accessory anchor residue at P1 for optimal binding of nonamer peptides.
حسب: Griffin, T, وآخرون
منشور في: (1997)

An HLA-B35-restricted epitope modified at an anchor residue results in an antagonist peptide.
حسب: Dong, T, وآخرون
منشور في: (1996)

Significance of the six peptide-binding pockets of HLA-A2.1 in influenza A matrix peptide-specific cytotoxic T-lymphocyte reactivity.
حسب: Matsui, M, وآخرون
منشور في: (1994)

Identification of T cell receptor recognition residues for a viral peptide presented by HLA B27.
حسب: Bowness, P, وآخرون
منشور في: (1994)

The solvent-inaccessible Cys67 residue of HLA-B27 contributes to T cell recognition of HLA-B27/peptide complexes.
حسب: Appel, H, وآخرون
منشور في: (2004)

IDENTIFICATION OF T-CELL RECEPTOR RECOGNITION RESIDUES FOR AN INFLUENZA-VIRUS NONAMER PEPTIDE EPITOPE PRESENTED BY HLA B27
حسب: Bowness, P, وآخرون
منشور في: (1995)

Interaction of HLA-B27 homodimers with KIR3DL1 and KIR3DL2, unlike HLA-B27 heterotrimers, is independent of the sequence of bound peptide.
حسب: Kollnberger, S, وآخرون
منشور في: (2007)

Misfolding of HLA-B27 as a result of its B pocket suggests a novel mechanism for its role in susceptibility to spondyloarthropathies.
حسب: Mear, J, وآخرون
منشور في: (1999)

HLA class I typing by PCR: HLA-B27 and an African B27 subtype.
حسب: Hill, A, وآخرون
منشور في: (1991)

The role of HLA-B27 in spondyloarthritis.
حسب: Allen, R, وآخرون
منشور في: (1999)

HLA-B27-RESTRICTED PEPTIDE PRESENTATION TO CYTOTOXIC T-CELLS IN REACTIVE ARTHRITIS
حسب: Bowness, P, وآخرون
منشور في: (1995)

Expression of aberrant HLA-B27 molecules is dependent on B27 dosage and peptide supply.
حسب: McHugh, K, وآخرون
منشور في: (2014)

ERAP2 is associated with ankylosing spondylitis in HLA-B27-positive and HLA-B27-negative patients.
حسب: Robinson, P, وآخرون
منشور في: (2015)

HLA-B27
حسب: Bowness, P
منشور في: (2015)

HOMOCYSTEINE AND HLA B27-ASSOCIATED ARTHROPATHIES
حسب: Gao, X, وآخرون
منشور في: (1995)

HLA-B27 transgenic rodents express cell surface HLA-B27 heavy chain homodimers and multimers.
حسب: Bowness, P, وآخرون
منشور في: (2003)

Functional characterisation of HLA-B27 in autoimmunity
حسب: Kollnberger, S, وآخرون
منشور في: (2001)

Identification of HLA-B27-restricted peptides from the Chlamydia trachomatis proteome with possible relevance to HLA-B27-associated diseases.
حسب: Kuon, W, وآخرون
منشور في: (2001)

Autoimmunity-associated T cell receptors recognize HLA-B*27-bound peptides
حسب: Yang, X, وآخرون
منشور في: (2022)

Residue Cys-67 of HLA-B27 contributes to antigen presentation and T cell recognition of HLA-B27 restricted T cell epitopes.
حسب: Appel, H, وآخرون
منشور في: (2004)

IMMUNOGENIC HIV VARIANT PEPTIDES THAT BIND TO HLA-B8 BUT FAIL TO STIMULATE CYTOTOXIC T-LYMPHOCYTE RESPONSES
حسب: Mcadam, S, وآخرون
منشور في: (1995)

HLA-B27: natural function and pathogenic role in spondyloarthritis.
حسب: McMichael, A, وآخرون
منشور في: (2002)

A cluster of mutations in HLA-A2 alpha 2 helix abolishes peptide recognition by T cells.
حسب: Moots, R, وآخرون
منشور في: (1991)

Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility.
حسب: Evans, D, وآخرون
منشور في: (2011)

Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility
حسب: Evans, D, وآخرون
منشور في: (2011)

The KIRs of HLA-B27: the role of HLA-B27 KIR interactions in infection and spondyloarthritis
حسب: Wong-Baeza, I, وآخرون
منشور في: (2011)

Identification and characterisation of cell surface HLA-B27 homodimers
حسب: Bird, L, وآخرون
منشور في: (2002)

Pathogen-derived HLA-E bound epitopes reveal broad primary anchor pocket tolerability and conformationally malleable peptide binding
حسب: Walters, L, وآخرون
منشور في: (2018)

The human major histocompatibility complex class Ib molecule HLA-E binds signal sequence-derived peptides with primary anchor residues at positions 2 and 9.
حسب: Braud, V, وآخرون
منشور في: (1997)

Importance of a conserved TCR J alpha-encoded tyrosine for T cell recognition of an HLA B27/peptide complex.
حسب: Bowness, P, وآخرون
منشور في: (1998)

Genotyping HLA-B27 in spondyloarthropathies.
حسب: Brown, M, وآخرون
منشور في: (1998)

HLA-B27 heavy chain homodimers are expressed in HLA-B27 transgenic rodent models of spondyloarthritis and are ligands for paired Ig-like receptors.
حسب: Kollnberger, S, وآخرون
منشور في: (2004)

Genetic modulation of antigen presentation by HLA-B27 molecules.
حسب: Pazmany, L, وآخرون
منشور في: (1992)

Analysis of mutant HLA-A2 molecules. Differential effects on peptide binding and CTL recognition.
حسب: Tussey, L, وآخرون
منشور في: (1994)

Evidence of widespread binding of HLA class I molecules to peptides.
حسب: Frelinger, J, وآخرون
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