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Glucose control in UKPDS type...
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Glucose control in UKPDS type 2 diabetic patients with and without autoantibodies to islet-cell cytoplasm and glutamic acid decarboxylase.
Dades bibliogràfiques
Autors principals:
Mehta, Z
,
Manley, S
,
Zimmett, P
,
Bottazzo, G
,
Cull, C
,
Holman, R
,
Turner, R
,
Grp, U
Format:
Journal article
Publicat:
1999
Fons
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UKPDS 25: autoantibodies to islet-cell cytoplasm and glutamic acid decarboxylase for prediction of insulin requirement in type 2 diabetes
per: Turner, R, et al.
Publicat: (1997)
UKPDS 25: autoantibodies to islet-cell cytoplasm and glutamic acid decarboxylase for prediction of insulin requirement in type 2 diabetes. UK Prospective Diabetes Study Group.
per: Turner, R, et al.
Publicat: (1997)
Islet autoantibodies in clinically diagnosed type 2 diabetes: prevalence and relationship with metabolic control (UKPDS 70).
per: Davis, T, et al.
Publicat: (2005)
Relationship of autoantibodies to glutamic acid decarboxylase (GADA) to deterioration of glycamic control assessed by therapy progression in latent autoimmune diabetes in adults (LADA) in the UKPDS
per: Clark, A, et al.
Publicat: (2005)
Plasma lipids and hypoglycaemic therapies over 6 years in Type 2 diabetic patients in the UKPDS
per: Cull, C, et al.
Publicat: (1999)