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Glucose control in UKPDS type 2 diabetic patients with and without autoantibodies to islet-cell cytoplasm and glutamic acid decarboxylase.

Glucose control in UKPDS type 2 diabetic patients with and without autoantibodies to islet-cell cytoplasm and glutamic acid decarboxylase.

Xehetasun bibliografikoak
Egile Nagusiak: Mehta, Z, Manley, S, Zimmett, P, Bottazzo, G, Cull, C, Holman, R, Turner, R, Grp, U
Formatua: Journal article
Argitaratua: 1999
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Antzeko izenburuak

  • UKPDS 25: autoantibodies to islet-cell cytoplasm and glutamic acid decarboxylase for prediction of insulin requirement in type 2 diabetes
    nork: Turner, R, et al.
    Argitaratua: (1997)
  • UKPDS 25: autoantibodies to islet-cell cytoplasm and glutamic acid decarboxylase for prediction of insulin requirement in type 2 diabetes. UK Prospective Diabetes Study Group.
    nork: Turner, R, et al.
    Argitaratua: (1997)
  • Islet autoantibodies in clinically diagnosed type 2 diabetes: prevalence and relationship with metabolic control (UKPDS 70).
    nork: Davis, T, et al.
    Argitaratua: (2005)
  • Relationship of autoantibodies to glutamic acid decarboxylase (GADA) to deterioration of glycamic control assessed by therapy progression in latent autoimmune diabetes in adults (LADA) in the UKPDS
    nork: Clark, A, et al.
    Argitaratua: (2005)
  • Plasma lipids and hypoglycaemic therapies over 6 years in Type 2 diabetic patients in the UKPDS
    nork: Cull, C, et al.
    Argitaratua: (1999)

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