Mechanical effects of human cardiac troponin C mutation Gly159Asp in exchanged rabbit psoas fibres

Mechanical effects of human cardiac troponin C mutation Gly159Asp in exchanged rabbit psoas fibres

מידע ביבליוגרפי
Main Authors:	Preston, L, Lipscomb, S, Robinson, P, Watkins, H, Redwood, C, Mogensen, J, Ashley, C
פורמט:	Conference item
יצא לאור:	2004

פריטים דומים

Functional effects of the DCM mutant Gly159Asp troponin C in skinned muscle fibres.
מאת: Preston, L, et al.
יצא לאור: (2007)

DCM troponin C mutant Gly159Asp blunts the response to troponin phosphorylation.
מאת: Preston, L, et al.
יצא לאור: (2007)

Mutations in fast skeletal troponin I, troponin T, and beta-tropomyosin that cause distal arthrogryposis all increase contractile function.
מאת: Robinson, P, et al.
יצא לאור: (2007)

A revised method of troponin exchange in permeabilised cardiac trabeculae using vanadate: functional consequences of a HCM-causing mutation in troponin I.
מאת: Preston, L, et al.
יצא לאור: (2006)

Effects of hypertrophic cardiomyopathy mutations in troponin I on contractility of skinned cardiac muscle fibres
מאת: Burton, D, et al.
יצא לאור: (2002)

Effects of troponin C isoform on the action of the cardiotonic agent EMD 57033.
מאת: Lipscomb, S, et al.
יצא לאור: (2005)

Effect of hypertrophic cardiomyopathy mutations in human cardiac muscle alpha -tropomyosin (Asp175Asn and Glu180Gly) on the regulatory properties of human cardiac troponin determined by in vitro motility assay.
מאת: Bing, W, et al.
יצא לאור: (2000)

Dilated cardiomyopathy causing mutations in cardiac troponin T (TNT), cardiac troponin C (TNC), and alpha tropomyosin (TM) reduce CA(2+) sensitivity and maximum calcium activation
מאת: Redwood, C, et al.
יצא לאור: (2004)

Structure-function relationships of eight mutations in troponin and tropomyosin that cause dilated cardiomyopathy
מאת: Mirza, M, et al.
יצא לאור: (2004)

The effect of HCM mutations in troponin T and troponin I on the calcium binding of human cardiac troponin
מאת: Redwood, C, et al.
יצא לאור: (2001)

Using fluorescence emission spectroscopy to asses the Ca2+ affinity of reconstituted troponin and thin filaments containing cardiomyopathy causing mutations of troponin and alpha-tropomyosin
מאת: Robinson, P, et al.
יצא לאור: (2007)

CD14 C-159T and toll-like receptor 4 Asp299Gly polymorphisms in surviving meningococcal disease patients.
מאת: Ariane Biebl, et al.
יצא לאור: (2009-10-01)

The effect of the Asp175Asn and Glu180Gly TPM1 mutations on actin-myosin interaction during the ATPase cycle.
מאת: Rysev, N, et al.
יצא לאור: (2012)

Hypertrophic and dilated cardiomyopathycausing mutations of troponin and alpha-tropomyosin have opposite effects on thin filament calcium binding
מאת: Robinson, P, et al.
יצא לאור: (2007)

Investigating the Effect of Cardiomyopathy-Causing Mutations in Cardiac Troponin-T on Calcium Buffering In Situ
מאת: Robinson, P, et al.
יצא לאור: (2010)

Functional effects of mutations in troponin T and tropomyosin which cause dilated cardiomyopathy
מאת: Robinson, P, et al.
יצא לאור: (2003)

Altered inhibitory and troponin C-binding activities in cardiac troponin I mutants known to cause hypertrophic cardiomyopathy
מאת: Elliott, K, et al.
יצא לאור: (2000)

Mutations in fast skeletal troponin I, troponin T and beta-tropomyosin that cause distal arthrogryposis all enhance thin filament activation
מאת: Robinson, P, et al.
יצא לאור: (2005)

Dilated and hypertrophic cardiomyopathy mutations in troponin and alpha-tropomyosin have opposing effects on the calcium affinity of cardiac thin filaments.
מאת: Robinson, P, et al.
יצא לאור: (2007)

Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathy.
מאת: Mogensen, J, et al.
יצא לאור: (2004)

Comparison of the functional effects of mutations in troponin T which cause dilated and hypertrophic cardiomyopathies
מאת: Robinson, P, et al.
יצא לאור: (2002)

Two mutations in human cardiac troponin I known to cause hypertrophic cardiomyopathy have different effects on cardiac muscle contraction
מאת: Burton, D, et al.
יצא לאור: (2000)

Investigation of troponin I mutations causing familial hypertrophic cardiomyopathy
מאת: Abdulrazzak, H, et al.
יצא לאור: (2001)

Bisphosphonate-functionalized cyclic Arg-Gly-Asp peptidomimetics
מאת: Carmelo Drago, et al.
יצא לאור: (2012-10-01)

Hypertrophic cardiomyopathy-causing Asp175asn and Glu180gly Tpm1 mutations shift tropomyosin strands further towards the open position during the ATPase cycle
מאת: Borovikov, Y, et al.
יצא לאור: (2011)

Hypertrophic cardiomyopathy-causing Asp175asn and Glu180gly Tpm1 mutations shift tropomyosin strands further towards the open position during the ATPase cycle.
מאת: Borovikov, Y, et al.
יצא לאור: (2011)

Tethering of Gly-Arg-Gly-Asp-Ser-Pro-Lys Peptides on Mg-Doped Hydroxyapatite
מאת: Alessandro Pistone, et al.
יצא לאור: (2017-02-01)

Two mutations in troponin I that cause hypertrophic cardiomyopathy have contrasting effects on cardiac muscle contractility.
מאת: Burton, D, et al.
יצא לאור: (2002)

Altered regulatory properties of human cardiac troponin I mutants that cause hypertrophic cardiomyopathy.
מאת: Elliott, K, et al.
יצא לאור: (2000)

Characterizations and molecular docking mechanism of the interactions between peptide FDGDF (Phe-Asp-Gly-Asp-Phe) and SOD enzyme
מאת: C.H.E.N. Wen-Tao, et al.
יצא לאור: (2024-01-01)

A troponin I mutant known to cause hypertrophic cardiomyopathy shows reduced inhibition of Ca2+ independent tension
מאת: Burton, D, et al.
יצא לאור: (2000)

Identification and functional analysis of cardiac troponin I as a novel disease gene in autosomal dominant familial dilated cardiomyopathy
מאת: Carballo, S, et al.
יצא לאור: (2006)

Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy.
מאת: Robinson, P, et al.
יצא לאור: (2002)

Cardiac troponin I is a potential novel substrate for AMP-activated protein kinase
מאת: Oliveira, S, et al.
יצא לאור: (2007)

An Asp7Gly substitution in PPARG is associated with decreased transcriptional activation activity.
מאת: Liushuai Hua, et al.
יצא לאור: (2014-01-01)

Effect of mutation Arg91Gly on the thermal stability of β-tropomyosin
מאת: Nevzorov, I, et al.
יצא לאור: (2008)

Histomorphometry of Ossification in Functionalised Ceramics with Tripeptide Arg-Gly-Asp (RGD): An In Vivo Study
מאת: Filippo Migliorini, et al.
יצא לאור: (2022-05-01)

A single amino acid (Asp159) from the dog prion protein suppresses the toxicity of the mouse prion protein in Drosophila
מאת: J. Sanchez-Garcia, et al.
יצא לאור: (2016-11-01)

Association of IRS1 (Gly972Arg) and IRS2 (Gly1057Asp) genes polymorphisms with OSA and NAFLD in Asian Indians.
מאת: Surya Prakash Bhatt, et al.
יצא לאור: (2021-01-01)

CDH1 ASP187GLY MUTATION IMPAIRS APC/C ACTIVITY LEADING TO PFKFB3 STABILIZATION
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