Interaction of HLA-B27 homodimers with KIR3DL1 and KIR3DL2, unlike HLA-B27 heterotrimers, is independent of the sequence of bound peptide.

Interaction of HLA-B27 homodimers with KIR3DL1 and KIR3DL2, unlike HLA-B27 heterotrimers, is independent of the sequence of bound peptide.

HLA-B27 can form beta-2 microglobulin (beta2m)-associated heterotrimers (HLA-B27) and beta2m-free homodimers (B27(2)). Here, we study the role of complexed peptide in the interaction of these forms of B27 with the killer cell immunoglobulin (Ig)-like receptors KIR3DL1 and KIR3DL2 and with Ig-like tr...

وصف كامل

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Kollnberger, S, Chan, A, Sun, M, Chen, L, Wright, C, di Gleria, K, McMichael, A, Bowness, P
التنسيق:	Journal article
اللغة:	English
منشور في:	2007

مواد مشابهة

HLA-B27 homodimers differ in their interactions with LILRB1, LILRB2, KIR3DL1 and KIR3DL2 compared to HLA-B27 heterotrimers
حسب: Giles, J, وآخرون
منشور في: (2010)

Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis.
حسب: Bowness, P, وآخرون
منشور في: (2011)

THE ROLE OF HLA-B27 KIR3DL2 INTERACTIONS IN THE PATHOGENESIS OF SPONDYLOARTHRITIS
حسب: Kollnberger, S, وآخرون
منشور في: (2012)

The KIRs of HLA-B27: the role of HLA-B27 KIR interactions in infection and spondyloarthritis
حسب: Wong-Baeza, I, وآخرون
منشور في: (2011)

Molecular determinants for KIR3DL2 binding to HLA-B27 and other HLA-class I ligands
حسب: Hatano, H, وآخرون
منشور في: (2013)

HLA-B27 and KIR3DL2 Interactions Promote The Differentiation and Survival Of Proinflammatory T Cells In Spondyloarthritis
حسب: Ridley, A, وآخرون
منشور في: (2013)

KIR3DL2 INTERACTION WITH HLA-B27 PROMOTES THE SURVIVAL OF TH17 CELLS IN ANKYLOSING SPONDYLITIS
حسب: Kollnberger, S, وآخرون
منشور في: (2010)

KIR3DL2 INTERACTION WITH HLA-B27 PROMOTES THE SURVIVAL OF TH17 CELLS IN ANKYLOSING SPONDYLITIS
حسب: Kollnberger, S, وآخرون
منشور في: (2010)

Crystal structures and KIR3DL1 recognition of three immunodominant viral peptides complexed to HLA-B*2705.
حسب: Stewart-Jones, G, وآخرون
منشور في: (2005)

The arthritis-associated HLA-B*27:05 allele forms more cell surface B27 dimer and free heavy chain ligands for KIR3DL2 than HLA-B*27:09.
حسب: Cauli, A, وآخرون
منشور في: (2013)

THE ARTHRITIS-ASSOCIATED HLA-B(star)27:05 FORMS MORE CELL SURFACE B27 DIMER AND FREE HEAVY CHAIN LIGANDS FOR KIR3DL2 AND LILRB2 IMMUNORECEPTORS THAN HLA(star)B27:09
حسب: Shaw, J, وآخرون
منشور في: (2013)

Identification and characterisation of cell surface HLA-B27 homodimers
حسب: Bird, L, وآخرون
منشور في: (2002)

HLA-B27 transgenic rodents express cell surface HLA-B27 heavy chain homodimers and multimers.
حسب: Bowness, P, وآخرون
منشور في: (2003)

HLA-B27 FORMS HEAVY CHAIN HOMODIMERS IN VITRO AND IN VIVO THAT BIND TO CELLULAR RECEPTORS INCLUDING LILR AND KIR FAMILY MEMBERS
حسب: McHugh, K, وآخرون
منشور في: (2010)

KIR3DL2 Binds to HLA-B27 Dimers and Free H Chains More Strongly than Other HLA Class I and Promotes the Expansion of T Cells in Ankylosing Spondylitis.
حسب: Wong-Baeza, I, وآخرون
منشور في: (2013)

KIR3DL2 binds to HLA-B27 dimers and free H chains more strongly than other HLA class I and promotes the expansion of T cells in ankylosing spondylitis.
حسب: Wong-Baeza, I, وآخرون
منشور في: (2013)

Cell-surface expression and immune receptor recognition of HLA-B27 homodimers.
حسب: Kollnberger, S, وآخرون
منشور في: (2002)

Functional characterisation of HLA-B27 in autoimmunity
حسب: Kollnberger, S, وآخرون
منشور في: (2001)

Recognition of HLA-A3 and HLA-A11 by KIR3DL2 is peptide-specific.
حسب: Hansasuta, P, وآخرون
منشور في: (2004)

TARGETING MONOCYTE-EXPRESSED HLA-B27 HOMODIMERS IN ANKYLOSING SPONDYLITIS
حسب: Bowness, P, وآخرون
منشور في: (2010)

TARGETING MONOCYTE-EXPRESSED HLA-B27 HOMODIMERS IN ANKYLOSING SPONDYLITIS
حسب: Bowness, P, وآخرون
منشور في: (2010)

HLA-B*2709 FORMS LESS CELL SURFACE B27 DIMER AND FREE HEAVY CHAIN LIGANDS FOR KIR3DL2 AND LILRB2 IMMUNORECEPTORS THAN ARTHRITIS-ASSOCIATED HLA-B*2705
حسب: Shaw, J, وآخرون
منشور في: (2012)

HLA-B27 heavy chain homodimers are expressed in HLA-B27 transgenic rodent models of spondyloarthritis and are ligands for paired Ig-like receptors.
حسب: Kollnberger, S, وآخرون
منشور في: (2004)

Cutting Edge: Allele-specific and peptide-dependent interactions between KIR3DL1 and HLA-A and HLA-B.
حسب: Thananchai, H, وآخرون
منشور في: (2007)

Cutting edge: Allele-specific and peptide-dependent interactions between KIR3DL1 and HLA-A and HLA-B
حسب: Thananchai, H, وآخرون
منشور في: (2007)

HLA-B-27 heavy chain homodimer formation and natural killer cell family interaction - comparison of two HLA-B-27 subtypes in vitro
حسب: Giles, J, وآخرون
منشور في: (2008)

HLA-B27
حسب: Bowness, P
منشور في: (2015)

Lymphoblastoid cells express HLA-B27 homodimers both intracellularly and at the cell surface following endosomal recycling.
حسب: Bird, L, وآخرون
منشور في: (2003)

The pathogenesis of spondyloarthritides - HLA-B-27 homodimer expression on human monocyte/macrophage cell lines
حسب: McHugh, K, وآخرون
منشور في: (2008)

Inhibiting HLA-B27 homodimer-driven immune cell inflammation in spondylarthritis.
حسب: Payeli, S, وآخرون
منشور في: (2012)

Molecular Characterisation Of The Killer Cell Immunoglobulin-Like Receptor 3DL2 Binding To Aberrant HLA-B27 Heavy Chains In Spondyloarthritis
حسب: Hatano, H, وآخرون
منشور في: (2013)

The role of HLA-B27 in spondyloarthritis.
حسب: Allen, R, وآخرون
منشور في: (1999)

HLA B27 can form heavy chain homodimers in vivo and in vivo, possible role of cys 67.
حسب: Bowness, P, وآخرون
منشور في: (1998)

EXPRESSION OF HLA-B27 HEAVY CHAIN FORMS IN ANKYLOSING SPONDYLITIS AND HLA-B27 POSITIVE CELL LINES
حسب: Rysnik, O, وآخرون
منشور في: (2012)

HLA-B27 homodimers and free H chains are stronger ligands for leukocyte Ig-like receptor B2 than classical HLA class I.
حسب: Giles, J, وآخرون
منشور في: (2012)

The role of cell surface HLA-B27 heavy chain homodimer interactions with NK receptors in the pathogenesis of spondyloarthritis
حسب: Kollnberger, S
منشور في: (2005)

Th17 cells expressing KIR3DL2+are increased in ankylosing spondylitis
حسب: Ridley, A, وآخرون
منشور في: (2010)

No association of KIR3DL1 or KIR3DS1 or their alleles with ankylosing spondylitis.
حسب: McCappin, J, وآخرون
منشور في: (2010)

Activation-induced killer cell immunoglobulin-like receptor 3DL2 binding to HLA-B27 licenses pathogenic T cell differentiation in spondyloarthritis
حسب: Ridley, A, وآخرون
منشور في: (2016)

HLA class I typing by PCR: HLA-B27 and an African B27 subtype.
حسب: Hill, A, وآخرون
منشور في: (1991)