Sulphonylurea therapy over six years does not delay progression to diabetes

Sulphonylurea therapy over six years does not delay progression to diabetes

Manylion Llyfryddiaeth
Prif Awduron:	Herlihy, O, Morris, R, Karunakaran, S, Holman, R, Grp, F
Fformat:	Journal article
Cyhoeddwyd:	2000

Eitemau Tebyg

Efficacy over six years of sulphonylurea plus insulin therapy in type 2 diabetic patients in the UKPDS
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Cyhoeddwyd: (2000)

Reinforced healthy living advice over six years does not delay progression to diabetes
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UKPDS 26: Sulphonylurea failure in non-insulin-dependent diabetic patients over six years
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Sulphonylurea failure in type 2 diabetes: treatment with a basal insulin supplement.
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Sulphonylureas and hypoglycaemia.
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UKPDS 18: Estimated dietary intake in type 2 diabetic patients randomly allocated to diet, sulphonylurea or insulin therapy
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This house believes that sulphonylureas should not be used routinely as second-line treatments for patients with type 2 diabetes
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Sulphonylureas in the management of type 2 diabetes: To be or not to be?
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Risk factors predicting progression of diabetic retinopathy over six years in 1579 NIDDM patients
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UKPDS 18: estimated dietary intake in type 2 diabetic patients randomly allocated to diet, sulphonylurea or insulin therapy. UK Prospective Diabetes Study Group.
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Conventional reinforced healthy living advice over three years does not delay transition to diabetes in subjects with raised fasting glucose levels.
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Possible prevention of type 2 diabetes with acarbose or metformin over three years
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Plasma lipids and hypoglycaemic therapies over 6 years in Type 2 diabetic patients in the UKPDS
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New uses for old drugs: neonatal diabetes and sulphonylureas.
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Neonatal Diabetes Caused by Activating Mutations in the Sulphonylurea Receptor
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Post-prandial amylin concentrations are increased by sulphonylurea, but unchanged by basal insulin, in NIDDM
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Extent of satisfaction with tablets and food-timing in sulphonylurea-treated diabetes.
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Permanent neonatal diabetes: combining sulphonylureas with insulin may be an effective treatment
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Pharmacogenetics in Type 2 diabetes: variation in TCF7L2 influences response to sulphonylureas
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Factors influencing utilization of different types of sulphonylureas amongst type 2 diabetes patients
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Sulphonylureas and the rise and fall of beta-cell function
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Glycemic control with diet, sulfonylurea, metformin, or insulin in patients with type 2 diabetes mellitus - Progressive requirement for multiple therapies (UKPDS 49)
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The Fasting Hyperglycaemia Study: III. Randomized controlled trial of sulfonylurea therapy in subjects with increased but not diabetic fasting plasma glucose.
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Identification of First Case of Neonatal Diabetes in Singapore and Successful Conversion from Insulin to Sulphonylurea
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Phentolamine block of ATP-sensitive K+ channels does not involve interaction with the sulphonylurea receptor, SUR1.
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Interactions between nicorandil and sulphonylureas: a structural basis.
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Sulphonylurea responsive monogenic diabetes in an Insulin treated 8 - year old child in West Africa; of more than academic interest and one of many?
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Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33)
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Microvascular and macrovascular disease in subjects with increased but not diabetic fasting glucose levels
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Promiscuous coupling between the sulphonylurea receptor and inwardly rectifying potassium channels.
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Homozygous hypomorphic HNF1A alleles are a novel cause of young-onset diabetes and result in sulphonylurea sensitive diabetes
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Effects of protein phosphorylation on the sulphonylurea receptor of the pancreatic beta-cell.
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Binding of sulphonylureas to plasma proteins – a KATP channel perspective
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Alternative MDR splicing is required for direct potentiation of exocytosis by sulphonylureas
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Binding of sulphonylureas to plasma proteins - A KATP channel perspective.
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Successful transition to sulphonylurea therapy from insulin in a child with permanent neonatal diabetes due to a KCNJ11 gene mutation
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RANDOMIZED CONTROLLED TRIAL OF SULFONYLUREA THERAPY IN 227 SUBJECTS WITH PERSISTENT FASTING HYPERGLYCEMIA
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A heterozygous activating mutation in the sulphonylurea receptor SUR1 (ABCC8) causes neonatal diabetes.
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Differential interactions of nateglinide and repaglinide on the human beta-cell sulphonylurea receptor 1.
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