No evidence for mutations in a putative beta-cell ATP-sensitive K+ channel subunit in MODY, NIDDM, or GDM.
The beta-cell ATP-sensitive K+ (K-ATP) channel has a major role in glucose-induced insulin secretion. Screening the entire coding sequence of the gene for a putative beta-cell K-ATP channel subunit, K-ATP2, with single-strand conformation polymorphism did not show any mutations associated with diabe...
Những tác giả chính: | Zhang, Y, Warren-Perry, M, Sakura, H, Adelman, J, Stoffel, M, Bell, G, Ashcroft, F, Turner, R |
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Định dạng: | Journal article |
Ngôn ngữ: | English |
Được phát hành: |
1995
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Những quyển sách tương tự
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NO EVIDENCE FOR MUTATIONS IN THE BETA-CELL ATP-SENSITIVE POTASSIUM CHANNEL IN MODY, NIDDM OR GESTATIONAL DIABETES
Bằng: Zhang, Y, et al.
Được phát hành: (1995) -
No evidence for mutations in a putative subunit of the beta-cell ATP-sensitive potassium channel (K-ATP channel) in Japanese NIDDM patients.
Bằng: Yasuda, K, et al.
Được phát hành: (1995) -
Screening for mutations in the Kir6.2 subunit of the beta-cell K-ATP channel
Bằng: Sakura, H, et al.
Được phát hành: (1996) -
Characterization and variation of a human inwardly-rectifying-K-channel gene (KCNJ6): a putative ATP-sensitive K-channel subunit.
Bằng: Sakura, H, et al.
Được phát hành: (1995) -
Sequence variations in the human Kir6.2 gene, a subunit of the beta-cell ATP-sensitive K-channel: no association with NIDDM in while Caucasian subjects or evidence of abnormal function when expressed in vitro.
Bằng: Sakura, H, et al.
Được phát hành: (1996)