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Aiddostahtton
  • miR-21 mediates hematopoietic...
  • Čujuhandieđut
  • Deakstadieđáhus
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  • Doalvvo čujuhusa
    • Doalvun: RefWorks
    • Doalvun: EndNoteWeb
    • Doalvun: EndNote
  • Bissovaš liŋka
miR-21 mediates hematopoietic suppression in MDS by activating TGF-beta signaling

miR-21 mediates hematopoietic suppression in MDS by activating TGF-beta signaling

Bibliográfalaš dieđut
Váldodahkkit: Bhagat, T, Zhou, L, Sokol, L, Kessel, R, Caceres, G, Gundabolu, K, Tamari, R, Gordon, S, Mantzaris, I, Jodlowski, T, Yu, Y, Jing, X, Polineni, R, Bhatia, K, Pellagatti, A, Boultwood, J, Kambhampati, S, Steidl, U, Stein, C, Ju, W, Liu, G, Kenny, P, List, A, Bitzer, M, Verma, A
Materiálatiipa: Journal article
Almmustuhtton: 2013
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Govvádus
Čoahkkáigeassu:

Geahča maid

  • miR-21 mediates hematopoietic suppression in MDS by activating TGF-β signaling.
    Dahkki: Bhagat, T, et al.
    Almmustuhtton: (2013)
  • Inhibition of the TGF-beta receptor I kinase promotes hematopoiesis in MDS.
    Dahkki: Zhou, L, et al.
    Almmustuhtton: (2008)
  • Reduced SMAD7 leads to overactivation of TGF-beta signaling in MDS that can be reversed by a specific inhibitor of TGF-beta receptor I kinase.
    Dahkki: Zhou, L, et al.
    Almmustuhtton: (2011)
  • The impact of spliceosome mutations in MDS
    Dahkki: Jacqueline Boultwood, et al.
    Almmustuhtton: (2019-06-01)
  • TP53 suppression promotes erythropoiesis in del(5q) MDS, suggesting a targeted therapeutic strategy in lenalidomide-resistant patients.
    Dahkki: Caceres, G, et al.
    Almmustuhtton: (2013)

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