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Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling

Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling

Dades bibliogràfiques
Autors principals: Antoniades, C, Shirodaria, C, Leeson, P, Van-Assche, T, Ratnatunga, C, Pillai, R, Tousoulis, D, Stefanadis, C, Refsurn, H, Channon, K
Format: Conference item
Publicat: 2008
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  • Genetic variability of MTHFR affects vascular redox and nitric oxide bioavailability in human vessels, by modifying eNOS coupling
    per: Antoniades, C, et al.
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  • Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
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  • Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
    per: Antoniades, C, et al.
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  • Plasma asymmetrical-dimethyl-arginine (ADMA) as a regulator of vascular redox and nitric oxide bioavailability in human vessels
    per: Antoniades, C, et al.
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  • The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
    per: Antoniades, C, et al.
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