Trisomy 21 expands the megakaryocyte-erythroid progenitor compartment in human fetal liver-implications for down syndrome AMKL.
Main Authors: | Tunstall-Pedoe, O, De la Fuente, J, Bennett, P, Fisk, N, Vyas, P, Roberts, I |
---|---|
Format: | Conference item |
Published: |
2006
|
Similar Items
-
Abnormalities in the myeloid progenitor compartment in Down syndrome fetal liver precede acquisition of GATA1 mutations.
by: Tunstall-Pedoe, O, et al.
Published: (2008) -
MicroRNAs in erythroid and megakaryocytic differentiation and megakaryocyte–erythroid progenitor lineage commitment
by: Zhang, L., et al.
Published: (2014) -
Single-cell profiling of human megakaryocyte-erythroid progenitors identifies distinct megakaryocyte and erythroid differentiation pathways
by: Psaila, B, et al.
Published: (2016) -
Severe TMD/AMKL with GATA1 mutation in a stillborn fetus with Down syndrome.
by: Heald, B, et al.
Published: (2007) -
Perturbation of fetal liver hematopoietic stem and progenitor cell development by trisomy 21.
by: Roy, A, et al.
Published: (2012)