Endothelial overexpression of Nox2 in transgenic mice potentiates vascular oxidative stress and the pressor response to angiotensin II

מידע ביבליוגרפי
Main Authors:	Bendall, J, Adlam, D, Rinze, R, Tatham, A, de Bono, J, Channon, K
פורמט:	Conference item
יצא לאור:	2007

Endothelial Nox2 overexpression potentiates vascular oxidative stress and hemodynamic response to angiotensin II: Studies in endothelial-targeted Nox2 transgenic mice
מאת: Bendall, J, et al.
יצא לאור: (2006)

Endothelial Nox2 overexpression potentiates vascular oxidative stress and hemodynamic response to angiotensin II: studies in endothelial-targeted Nox2 transgenic mice.
מאת: Bendall, J, et al.
יצא לאור: (2007)

Endothelial Nox2 overexpression potentiates vascular oxidative stress and hemodynamic response to angiotensin II: Studies in endothelial-targeted Nox2 transgenic mice (vol 100, pg 1016, 2007)
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יצא לאור: (2008)

Endothelial-specific Nox2 overexpression increases vascular superoxide and macrophage recruitment in ApoE⁻/⁻ mice.
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Endothelial specific Nox2 overexpression increases vascular superoxide production but does not alter atherosclerotic plaque or neointima formation in mice
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Targeted endothelial Nox2 over-expression potentiates angiotensin ii induced aortic dissection in mice
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ENDOTHELIAL SPECIFIC NOX2 OVER-EXPRESSION INCREASES SUSCEPTIBILITY TO ANGIOTENSIN II INDUCED AORTIC DISSECTION
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Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
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Relationships between nitric oxide-mediated endothelial function, eNOS coupling and blood pressure revealed by eNOS-GTP cyclohydrolase 1 double transgenic mice.
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Endothelial-specific Nox2 over-expression increases vascular superoxide production but does not alter atherosclerotic plaque or AngII-dependent aneurysm formation
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Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
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Endothelial tetrahydrobiopterin regulates eNOS coupling/uncoupling independent of vascular disease: Insights from targeted transgenic models
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Endothelial tetrahydrobiopterin (BH4) regulates eNOS coupling in vivo: Functional effects of BH4-eNOS stoichiometry in endothelial targeted transgenic mice
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The of endothelial cells in the regulation of the vascular response to Angiotensin II
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Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
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Increased eNOS coupling protects against vein graft accelerated atherosclerosis in ApoE-KO mice
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Endothelial overexpression of Nox2 in transgenic mice potentiates vascular oxidative stress and the pressor response to angiotensin II

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