Endothelial overexpression of Nox2 in transgenic mice potentiates vascular oxidative stress and the pressor response to angiotensin II

Chi tiết về thư mục
Những tác giả chính:	Bendall, J, Adlam, D, Rinze, R, Tatham, A, de Bono, J, Channon, K
Định dạng:	Conference item
Được phát hành:	2007

Endothelial Nox2 overexpression potentiates vascular oxidative stress and hemodynamic response to angiotensin II: Studies in endothelial-targeted Nox2 transgenic mice
Bằng: Bendall, J, et al.
Được phát hành: (2006)

Endothelial Nox2 overexpression potentiates vascular oxidative stress and hemodynamic response to angiotensin II: studies in endothelial-targeted Nox2 transgenic mice.
Bằng: Bendall, J, et al.
Được phát hành: (2007)

Endothelial Nox2 overexpression potentiates vascular oxidative stress and hemodynamic response to angiotensin II: Studies in endothelial-targeted Nox2 transgenic mice (vol 100, pg 1016, 2007)
Bằng: Bendall, J, et al.
Được phát hành: (2008)

Endothelial-specific Nox2 overexpression increases vascular superoxide and macrophage recruitment in ApoE⁻/⁻ mice.
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Được phát hành: (2012)

Endothelial specific Nox2 overexpression increases vascular superoxide production but does not alter atherosclerotic plaque or neointima formation in mice
Bằng: Douglas, G, et al.
Được phát hành: (2008)

Targeted endothelial Nox2 over-expression potentiates angiotensin ii induced aortic dissection in mice
Bằng: Fan, L, et al.
Được phát hành: (2010)

ENDOTHELIAL SPECIFIC NOX2 OVER-EXPRESSION INCREASES SUSCEPTIBILITY TO ANGIOTENSIN II INDUCED AORTIC DISSECTION
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Được phát hành: (2012)

Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
Bằng: Bendall, J, et al.
Được phát hành: (2005)

Relationships between nitric oxide-mediated endothelial function, eNOS coupling and blood pressure revealed by eNOS-GTP cyclohydrolase 1 double transgenic mice.
Bằng: Adlam, D, et al.
Được phát hành: (2007)

Endothelial-specific Nox2 over-expression increases vascular superoxide production but does not alter atherosclerotic plaque or AngII-dependent aneurysm formation
Bằng: Douglas, G, et al.
Được phát hành: (2010)

Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
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Được phát hành: (2004)

Increased endothelial tetrahydrobiopterin synthesis reduces vein graft accelerated atherosclerosis in apolipoprotein E-knockout mice
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Presence of atherosclerosis determines the vascular but not the cardiac effects of exercise training through endothelial NOS activity
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Endothelial tetrahydrobiopterin regulates eNOS coupling/uncoupling independent of vascular disease: Insights from targeted transgenic models
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Endotoxemia-related acute kidney injury in transgenic mice with endothelial overexpression of GTP cyclohydrolase-1.
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Tetrahydrobiopterin-dependent eNOS coupling determines vascular remodeling through accelerated endothelial regeneration
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Increased endothelial tetrahydrobiopterin synthesis by targeted transgenic GTP-cyclohydrolase I overexpression reduces endothelial dysfunction and atherosclerosis in ApoE-knockout mice.
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Endothelial tetrahydrobiopterin (BH4) regulates eNOS coupling in vivo: Functional effects of BH4-eNOS stoichiometry in endothelial targeted transgenic mice
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Inhibition of endothelial Nox2 activation by LMH001 protects mice from angiotensin II-induced vascular oxidative stress, hypertension and aortic aneurysm
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Vasoconstrictor and Pressor Effects of Des-Aspartate-Angiotensin I in Rat
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Increased endothelial tetrahydrobiopterin levels in GTPCH-transgenic mice
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Tetrahydrobiopterin (BH4) Improves Endothelial Cell Survival After Vascular Injury
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Differential contribution of Nox1, Nox2 and Nox4 to kidney vascular oxidative stress and endothelial dysfunction in obesity
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Increased endothelial tetrahydrobiopterin levels by GTPCH over expression in transgenic mice prevents diabetic endothelial dysfunction
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The of endothelial cells in the regulation of the vascular response to Angiotensin II
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Inducibility, but not stability, of atrial fibrillation is increased by NOX2 overexpression in mice
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CCR2-mediated antiinflammatory effects of endothelial tetrahydrobiopterin inhibit vascular injury-induced accelerated atherosclerosis.
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Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
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The relative contributions of cardiomyocytes versus endothelial cells to cardiac BH4 production; studies in novel cardiomyocyte- and endothelial-specific GTPCH I-deficient mice
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Increased eNOS coupling protects against vein graft accelerated atherosclerosis in ApoE-KO mice
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Endothelial cell tetrahydrobiopterin modulates sensitivity to Ang (Angiotensin) II–induced vascular remodeling, blood pressure, and abdominal aortic aneurysm
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Endothelial overexpression of Nox2 in transgenic mice potentiates vascular oxidative stress and the pressor response to angiotensin II

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