TNO evidence to support CTLA4 as a multiple sclerosis susceptibility gene

Bibliographic Details
Main Authors: Morrison, K, Dyment, D, Willer, C, Risch, N, Sadovnik, A, Ebers, G
Format: Journal article
Published: 2002
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author Morrison, K
Dyment, D
Willer, C
Risch, N
Sadovnik, A
Ebers, G
author_facet Morrison, K
Dyment, D
Willer, C
Risch, N
Sadovnik, A
Ebers, G
author_sort Morrison, K
collection OXFORD
description
first_indexed 2024-03-07T06:43:11Z
format Journal article
id oxford-uuid:f9fe34d1-68d6-43ca-84c8-adafc8930c94
institution University of Oxford
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publishDate 2002
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spelling oxford-uuid:f9fe34d1-68d6-43ca-84c8-adafc8930c942022-03-27T13:02:11ZTNO evidence to support CTLA4 as a multiple sclerosis susceptibility geneJournal articlehttp://purl.org/coar/resource_type/c_dcae04bcuuid:f9fe34d1-68d6-43ca-84c8-adafc8930c94Symplectic Elements at Oxford2002Morrison, KDyment, DWiller, CRisch, NSadovnik, AEbers, G
spellingShingle Morrison, K
Dyment, D
Willer, C
Risch, N
Sadovnik, A
Ebers, G
TNO evidence to support CTLA4 as a multiple sclerosis susceptibility gene
title TNO evidence to support CTLA4 as a multiple sclerosis susceptibility gene
title_full TNO evidence to support CTLA4 as a multiple sclerosis susceptibility gene
title_fullStr TNO evidence to support CTLA4 as a multiple sclerosis susceptibility gene
title_full_unstemmed TNO evidence to support CTLA4 as a multiple sclerosis susceptibility gene
title_short TNO evidence to support CTLA4 as a multiple sclerosis susceptibility gene
title_sort tno evidence to support ctla4 as a multiple sclerosis susceptibility gene
work_keys_str_mv AT morrisonk tnoevidencetosupportctla4asamultiplesclerosissusceptibilitygene
AT dymentd tnoevidencetosupportctla4asamultiplesclerosissusceptibilitygene
AT willerc tnoevidencetosupportctla4asamultiplesclerosissusceptibilitygene
AT rischn tnoevidencetosupportctla4asamultiplesclerosissusceptibilitygene
AT sadovnika tnoevidencetosupportctla4asamultiplesclerosissusceptibilitygene
AT ebersg tnoevidencetosupportctla4asamultiplesclerosissusceptibilitygene