Vascular endothelial growth factor C is increased in endometrium and promotes endothelial functions, vascular permeability and angiogenesis and growth of endometriosis
Endometriosis is an angiogenesis-dependent disease. Many studies demonstrated inhibition of angiogenesis leads to inhibition of endometriotic growth, however underlying mechanism is still not fully understood. Our previous study suggested vascular endothelial growth factor C (VEGF-C) as a target of...
المؤلفون الرئيسيون: | Xu, H, Zhang, T, Man, G, May, K, Becker, C, Davis, T, Kung, A, Birsner, A, D'Amato, R, Wong, A, Wang, C |
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التنسيق: | Journal article |
اللغة: | English |
منشور في: |
2013
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مواد مشابهة
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Vascular endothelial growth factor C is increased in endometrium and promotes endothelial functions, vascular permeability and angiogenesis and growth of endometriosis.
حسب: Xu, H, وآخرون
منشور في: (2013) -
Green tea epigallocatechin-3-gallate inhibits angiogenesis and suppresses vascular endothelial growth factor C/vascular endothelial growth factor receptor 2 expression and signaling in experimental endometriosis in vivo.
حسب: Xu, H, وآخرون
منشور في: (2011) -
Differential Suppression of Vascular Permeability and Corneal Angiogenesis by Nonsteroidal Anti-inflammatory Drugs
حسب: Pakneshan, P, وآخرون
منشور في: (2008) -
Anti-Vascular Endothelial Growth Factor Agents for Ocular Angiogenesis and Vascular Permeability
حسب: Toshiaki Kubota, وآخرون
منشور في: (2012-01-01) -
Expression of vascular endothelial growth factor a in the endometrium in uterine factor infertility
حسب: Liudmila M. Chuprinenko, وآخرون
منشور في: (2020-02-01)