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Nitric oxide Bioavailability i...
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Nitric oxide Bioavailability is preserved in diabetes by increased endothelial tetrahydrobiopterin synthesis in targeted GTP cyclohydrolase transgenic mice
Bibliografiske detaljer
Main Authors:
Alp, N
,
Mussa, S
,
Khoo, J
,
Cai, S
,
Guzik, T
,
Rockett, K
,
Channon, K
Format:
Conference item
Udgivet:
2003
Beholdninger
Beskrivelse
Lignende værker
Medarbejdervisning
Lignende værker
Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
af: Alp, N, et al.
Udgivet: (2003)
Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I over-expression
af: Alp, N, et al.
Udgivet: (2003)
Nitric oxide bioactivity in ApoE -/- mice is maintained by increased endothelial tetrahydrobiopterin synthesis via targeted GTP cyclohydrolase-1 over-expression
af: Alp, N, et al.
Udgivet: (2003)
Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
af: Bendall, J, et al.
Udgivet: (2004)
Increased endothelial tetrahydrobiopterin synthesis by targeted transgenic GTP-cyclohydrolase I overexpression reduces endothelial dysfunction and atherosclerosis in ApoE-knockout mice.
af: Alp, N, et al.
Udgivet: (2004)