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Oral Fibrosis and Oral Cancer: From Molecular Targets to Therapeutics
Published 2022-05-01Get full text
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E3 ligase STUB1 attenuates stemness and tumorigenicity of oral carcinoma cells via transglutaminase 2 regulation
Published 2020-10-01Subjects: “…Oral cancer…”
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LINC01296 promotes cancer stemness traits in oral carcinomas by sponging miR-143
Published 2023-04-01Subjects: Get full text
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Emerging Role of MicroRNA-200 Family in Dentistry
Published 2021-06-01Subjects: Get full text
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Butylidenephthalide Abrogates the Snail-Induced Cancer Stemness in Oral Carcinomas
Published 2022-05-01Subjects: Get full text
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miR-509 inhibits cancer stemness properties in oral carcinomas via directly targeting PlK1
Published 2022-04-01Subjects: Get full text
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MicroRNAs as Theranostics Targets in Oral Carcinoma Stem Cells
Published 2020-02-01Subjects: Get full text
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LINC00963 Promotes Cancer Stemness, Metastasis, and Drug Resistance in Head and Neck Carcinomas via ABCB5 Regulation
Published 2020-04-01“…Stemness phenotypes and the expression of CSCs markers in oral cancer cells transfected with sh-LINC00963 were examined. …”
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miR-1246 Targets CCNG2 to Enhance Cancer Stemness and Chemoresistance in Oral Carcinomas
Published 2018-08-01“…In the present work, we employed miRNAs RT-PCR analysis to evaluate the expression of miR-1246 in tumor tissues and oral cancer stem cells (OCSC). Stemness phenotypes, including self-renewal, migration, invasion, colony formation capacities, and in vivo oncogenicity of oral cancer cells following transfected with miR-1246 inhibitors or mimics were examined. …”
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miR-21 promotes the fibrotic properties in oral mucosa through targeting PDCD4
Published 2022-04-01“…Background/purpose: Oral submucous fibrosis (OSF) has been regarded as a premalignant disorder of oral cancer, and myofibroblasts are the main cells that are responsible for pathological fibrosis. …”
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Down-regulation of miR-29c promotes the progression of oral submucous fibrosis through targeting tropomyosin-1
Published 2022-06-01“…The expression of miR-29c is dysregulated in oral cancer, but its role in OSF has not been investigated. …”
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