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Changes in Reactivity In Vitro of CD4+CD25+ and CD4+CD25− T Cell Subsets in Transplant Tolerance
Published 2017-08-01“…To identify changes that may diagnose tolerance, changes in the patterns of proliferation of CD4+, CD4+CD25+, and CD4+CD25− T cells from DA rats tolerant to Piebald Virol Glaxo rat strain (PVG) cardiac allografts and from naïve DA rats were examined. …”
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62
First record of pigmentation disorder in the Fringe-lipped Bat Trachops cirrhosus (Spix, 1823) (Chiroptera: Phyllostomidae) from southeast Brazil
Published 2019-08-01“…Piebaldism is a genetic pigmentation disorder, which is caused by absence of melanocytes in parts of the skin and/or hair follicles, with eyes and claws normally pigmented. …”
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63
YY1 regulates melanocyte development and function by cooperating with MITF.
Published 2012-01-01“…YY1 cooperates with M-MITF in regulating the expression of piebaldism gene KIT and multiple additional pigmentation genes. …”
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64
YY1 Regulates Melanocyte Development and Function by Cooperating with MITF
Published 2012“…YY1 cooperates with M-MITF in regulating the expression of piebaldism gene KIT and multiple additional pigmentation genes. …”
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65
Analogs of human genetic skin disease in domesticated animals
Published 2017-09-01“…In addition, we will explore how animal diseases serve as a model to uncover the mechanisms of human disease.The genetic skin diseases we will review are pigmentary mosaicism, piebaldism, albinism, Griscelli syndrome, ectodermal dysplasias, Waardenburg syndrome, and mucinosis in both humans and domesticated animals.…”
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66
Identification of kit-ligand a as the Gene Responsible for the Medaka Pigment Cell Mutant few melanophore
Published 2020-01-01“…Our results may provide insight into the pathogenesis of c-Kit–related pigmentation disorders such as piebaldism in humans, and our kitlga knockout medaka may prove useful as a tool for drug screening.…”
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67
Naa12 compensates for Naa10 in mice in the amino-terminal acetylation pathway
Published 2021-08-01“…Rather Naa10 nulls display increased neonatal lethality, and the majority of surviving undersized mutants exhibit a combination of hydrocephaly, cardiac defects, homeotic anterior transformation, piebaldism, and urogenital anomalies. Naa12 is a previously unannotated Naa10-like paralog with NAT activity that genetically compensates for Naa10. …”
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68
Cellular and ultrastructural characterization of the grey-morph phenotype in southern right whales (Eubalaena australis).
Published 2017-01-01“…Grey morphism is distinct from piebaldism and albinism found in other species, which are genetic pigmentation conditions resulting from the local absence of melanocytes, or the inability to synthesize melanin, respectively.…”
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