Peptide inhibitors against influenza virus by Mohamed Ibrhaim, Mohamed Rajik, Yusoff, Khatijah

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Influenza viruses for next year's vaccines named

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The deadly dance of alveolar macrophages and influenza virus by Camille David, Charles Verney, Mustapha Si-Tahar, Antoine Guillon

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Roles of lncRNAs in influenza virus infection by Jing Wang, Shan Cen

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Influenza Virus-cell Fusion Inhibition Assay by Mayo Yasugi, Kazuyoshi Ikuta

“…Here we describe in vitro influenza virus-cell fusion inhibition assay. The infected cells expressing viral membrane protein, such as hemagglutinin (HA), on cellular surface are incubated with monoclonal antibodies targeting viral membrane protein. …”
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Unveiling the intersection: ferroptosis in influenza virus infection by Arash Letafati, Zahra Taghiabadi, Omid Salahi Ardekani, Simin Abbasi, Ali Qaraee Najafabadi, Negar Nayerain Jazi, Roben Soheili, Ramón Rodrigo, Jila Yavarian, Luciano Saso

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PAIVS: prediction of avian influenza virus subtype by Hyeon-Chun Park, Juyoun Shin, Sung-Min Cho, Shinseok Kang, Yeun-Jun Chung, Seung-Hyun Jung

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Corrigendum to “Influenza Virus Aerosols in the Air and Their Infectiousness” by Nikolai Nikitin, Ekaterina Petrova, Ekaterina Trifonova, Olga Karpova

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The effect of nonpharmaceutical interventions on influenza virus transmission by Danlei Chen, Danlei Chen, Ting Zhang, Simiao Chen, Xuanwen Ru, Qingyi Shao, Qingyi Shao, Qing Ye, Dongqing Cheng

Subjects: “…influenza virus…”
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Association of Influenza Virus Proteins with Membrane Rafts by Michael Veit, Bastian Thaa

“…Assembly and budding of influenza virus proceeds in the viral budozone, a domain in the plasma membrane with characteristics of cholesterol/sphingolipid-rich membrane rafts. …”
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Cellular and Molecular Immunity to Influenza Viruses and Vaccines by Jane Kasten-Jolly, David A. Lawrence

Subjects: “…influenza virus…”
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CD8⁺ T Cell Response to Influenza Virus by Doty, Daniel, S.M. Massachusetts Institute of Technology

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Learning evolutionary and virulence patterns of influenza viruses by Tan, Tosy Ying Jie

“…Although the classification results were not as ideal, we managed to conclude that the median lethal dosage has an influence in determining the virulence level of an influenza virus. It was also proven that the HA segments contains crucial information on the virulence of influenza viruses as well as shown that categorisation by host strains seems to produce better classification results. …”
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Predicting the antigenicity and reassortment probability of influenza viruses by Ong, Wenqi

“…The emergence of novel combinations of influenza virus strains has been the main cause of pandemics as it outpaces the development of flu vaccines to bring the spread of the influenza virus under control. …”
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A mechanism for the activation of the influenza virus transcriptase by Serna Martin, I, Hengrung, N, Renner, M, Sharps, J, Martinez Alonso, M, Masiulis, S, Grimes, J, Fodor, E

“…Influenza virus RNA polymerase (FluPol), a heterotrimer composed of PB1, PB2, and PA subunits (P3 in influenza C), performs both transcription and replication of the viral RNA genome. …”

The effect of influenza virus on the human oropharyngeal microbiome by Ramos-Sevillano, E, Wade, W, Mann, A, Gilbert, A, Lambkin-Williams, R, Killingley, B, Nguyen-Van-Tam, JS, Tang, CM

INTERACTION BETWEEN INFLUENZA-VIRUS AND HLA ANTIGENS by Hildreth, J, Mcmichael, A

HLA COMPLEX AND THE IMMUNE-RESPONSE TO INFLUENZA-VIRUS by Mcmichael, A, Weatherall, D

Recognition of influenza virus proteins by cytotoxic T lymphocytes. by Townsend, A, Bastin, J, Bodmer, H, Brownlee, G, Davey, J, Gotch, F, Gould, K, Jones, I, McMichael, A, Rothbard, J

“…Recombinant DNA techniques have been used to express the proteins of influenza virus individually. Target cells expressing single viral proteins were then used to identify the molecules recognized by cytotoxic T lymphocytes (CTLS). …”

The influenza virus panhandle is involved in the initiation of transcription. by Fodor, E, Pritlove, D, Brownlee, G

“…We propose a new model for the initiation of transcription which has implications for the mechanisms by which influenza virus transcription, replication, and polyadenylation may be regulated in the infected cell.…”