AHR Signaling Dampens Inflammatory Signature in Neonatal Skin γδ T Cells

AHR Signaling Dampens Inflammatory Signature in Neonatal Skin γδ T Cells

Background Aryl hydrocarbon receptor (AHR)-deficient mice do not support the expansion of dendritic epidermal T cells (DETC), a resident immune cell population in the murine epidermis, which immigrates from the fetal thymus to the skin around birth. Material and Methods In order to identify the gene...

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Bibliographic Details
Main Authors: Katja Merches, Alfonso Schiavi, Heike Weighardt, Swantje Steinwachs, Nadine Teichweyde, Irmgard Förster, Katrin Hochrath, Beatrix Schumak, Natascia Ventura, Patrick Petzsch, Karl Köhrer, Charlotte Esser
Format: Article
Language:English
Published: MDPI AG 2020-03-01
Series:International Journal of Molecular Sciences
Subjects:
γδ t cell
epidermis
aryl hydrocarbon receptor
Online Access:https://www.mdpi.com/1422-0067/21/6/2249

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https://www.mdpi.com/1422-0067/21/6/2249

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